Document Detail


Endogenous opioid peptides: comparative evaluation of their receptor affinities in the mouse brain.
MedLine Citation:
PMID:  6319879     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The affinities of certain endogenous opioid peptides and related sequences for mu, delta and k opiate receptors have been determined in membrane preparations from mouse brain. It was found that the KIs for the delta receptor changed very little when the sequence of the enkephalins was enlarged; on the contrary, the mu and especially the k activity were highly dependent not only on the specificity of the sequence but also on the length of the peptide. Most of the peptides have similar affinities for more than one receptor type. The enkephalins are the most selective for the delta receptor. beta-Endorphin, BAM-12P, BAM-22P, peptide E and dynorphin A display the best potency at the mu site. Dynorphin A (1-8), dynorphin A and dynorphin B are the most selective for the k site.
Authors:
J Garzón; P Sánchez-Blázquez; V Höllt; N M Lee; H H Loh
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Life sciences     Volume:  33 Suppl 1     ISSN:  0024-3205     ISO Abbreviation:  Life Sci.     Publication Date:  1983  
Date Detail:
Created Date:  1984-03-02     Completed Date:  1984-03-02     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0375521     Medline TA:  Life Sci     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  291-4     Citation Subset:  IM    
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MeSH Terms
Descriptor/Qualifier:
Animals
Brain / metabolism*
Endorphins / metabolism*
Kinetics
Mice
Receptors, Opioid / metabolism*
Receptors, Opioid, delta
Receptors, Opioid, kappa
Receptors, Opioid, mu
Grant Support
ID/Acronym/Agency:
DA-02643/DA/NIDA NIH HHS; FO-5TW-03080/TW/FIC NIH HHS
Chemical
Reg. No./Substance:
0/Endorphins; 0/Receptors, Opioid; 0/Receptors, Opioid, delta; 0/Receptors, Opioid, kappa; 0/Receptors, Opioid, mu

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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