Document Detail


Elevation of cytosolic calcium is sufficient to induce growth inhibition in a B cell lymphoma.
MedLine Citation:
PMID:  7691606     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Recently, we have described that anti-IgM antibodies profoundly inhibited the growth of BKS-2, an immature B cell lymphoma. In this report, we demonstrated that ionomycin alone at very low concentrations (20 nM) inhibited the growth of BKS-2 cells completely. The levels of intracellular Ca2+ induced by the inhibitory concentrations of ionomycin were comparable to those in anti-IgM-treated cells. The growth inhibition caused by ionomycin was reversed by phorbol 12-myristate 13-acetate and lipopolysaccharide. In addition, the immunosuppressants, cyclosporin A and FK506 conferred significant protection from the negative signal induced by ionomycin. However, either cyclosporin A, FK506 or lipopolysaccharide was not found to have direct effect on ionomycin-induced Ca2+ mobilization in BKS-2 cells. Also, ionomycin augmented the anti-IgM-induced growth arrest in these cells. Furthermore, BKS-2 cells that were exposed to anti-IgM or ionomycin underwent apoptosis as characterized by DNA fragmentation. Thus, the characteristics of growth inhibition induced by ionomycin and anti-IgM appeared to be similar in that phorbol 12-myristate 13-acetate, lipopolysaccharide, cyclosporin A and FK506 caused significant reversal from such negative signals and both ionomycin and anti-IgM induced apoptosis in these cells. Altogether, these results showed that the elevation of intracellular Ca2+ alone was sufficient to inhibit the growth of some B lymphoma cells.
Authors:
S Muthukkumar; V Udhayakumar; S Bondada
Related Documents :
10457356 - Translocation of marcks and reorganization of the cytoskeleton by pma correlates with t...
2297776 - Effect of 12-o-tetradecanoylphorbol-13-acetate on intercellular communication in variou...
6776326 - Differentiation of normal and cultured preneoplastic tracheal epithelial cells in rats:...
3435916 - Intercellular contacts and the organization of actin filaments in cultured epithelial f...
10494776 - Modulation of cell-associated plasminogen activation by stromelysin-1 (mmp-3).
8021216 - A statistical analysis of the formation of plasmid-free cells in populations of escheri...
Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  European journal of immunology     Volume:  23     ISSN:  0014-2980     ISO Abbreviation:  Eur. J. Immunol.     Publication Date:  1993 Oct 
Date Detail:
Created Date:  1993-11-12     Completed Date:  1993-11-12     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  1273201     Medline TA:  Eur J Immunol     Country:  GERMANY    
Other Details:
Languages:  eng     Pagination:  2419-26     Citation Subset:  IM    
Affiliation:
Department of Microbiology and Immunology, University of Kentucky, Lexington 40536-0230.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Antibodies, Anti-Idiotypic / pharmacology
Calcium / metabolism*
Cell Division / drug effects
Cyclosporine / pharmacology
Cytosol / metabolism
Immunoglobulin M
Ionomycin / pharmacology
Lipopolysaccharides / pharmacology
Lymphoma, B-Cell / drug therapy,  metabolism*,  pathology
Mice
Mice, Inbred CBA
Tacrolimus / pharmacology
Tetradecanoylphorbol Acetate / pharmacology
Grant Support
ID/Acronym/Agency:
AG 05731/AG/NIA NIH HHS; AI 21490/AI/NIAID NIH HHS; K04 AG 00422/AG/NIA NIH HHS
Chemical
Reg. No./Substance:
0/Antibodies, Anti-Idiotypic; 0/Immunoglobulin M; 0/Lipopolysaccharides; 109581-93-3/Tacrolimus; 16561-29-8/Tetradecanoylphorbol Acetate; 56092-81-0/Ionomycin; 59865-13-3/Cyclosporine; 7440-70-2/Calcium

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Preferential distribution of V beta 8.2-positive T cells in the central nervous system of rats with ...
Next Document:  The monoclonal antibody CZ-1 identifies a mouse CD45-associated epitope expressed on interleukin-2-r...