Document Detail


Elevated C1rC1sC1inh levels independently predict atherosclerotic coronary heart disease.
MedLine Citation:
PMID:  23174605     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
CONCLUSION: Activation of the classical complement pathway can be observed in angiographically proven coronary atherosclerosis. Elevated C1rC1sC1inh levels might represent an useful biomarker for coronary artery disease.
Authors:
Zsófia Horváth; Dorottya Csuka; Katarina Vargova; Andrea Kovács; Andrea Ágnes Molnár; Petra Gulácsi-Bárdos; Sarolta Leé; Lilian Varga; Róbert Gábor Kiss; István Préda; George Füst
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Publication Detail:
Type:  Evaluation Studies; Journal Article; Research Support, Non-U.S. Gov't     Date:  2012-11-20
Journal Detail:
Title:  Molecular immunology     Volume:  54     ISSN:  1872-9142     ISO Abbreviation:  Mol. Immunol.     Publication Date:  2013 May 
Date Detail:
Created Date:  2013-01-18     Completed Date:  2013-03-21     Revised Date:  2013-05-20    
Medline Journal Info:
Nlm Unique ID:  7905289     Medline TA:  Mol Immunol     Country:  England    
Other Details:
Languages:  eng     Pagination:  8-13     Citation Subset:  IM    
Copyright Information:
Copyright © 2012 Elsevier Ltd. All rights reserved.
Affiliation:
Research Group for Inflammation Biology and Immunogenomics of Hungarian Academy of Sciences and Semmelweis University, Róbert Károly krt. 44, 1134 Budapest, Hungary. horvzsofi@hotmail.com
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MeSH Terms
Descriptor/Qualifier:
Aged
Atherosclerosis / blood,  diagnosis*
Case-Control Studies
Cohort Studies
Complement Activation / physiology
Complement C1 / analysis*,  metabolism
Complement C1 Inactivator Proteins / analysis*,  metabolism
Complement C1r / analysis,  metabolism
Complement C1s / analysis,  metabolism
Coronary Artery Disease / blood,  diagnosis*
Female
Humans
Male
Middle Aged
Prognosis
Chemical
Reg. No./Substance:
0/Complement C1; 0/Complement C1 Inactivator Proteins; EC 3.4.21.41/Complement C1r; EC 3.4.21.42/Complement C1s

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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