Document Detail


Electromyographic evidence of subclinical myopathy in hypertrophic cardiomyopathy.
MedLine Citation:
PMID:  11102909     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Hypertrophic cardiomyopathy (HCM) is due to a number of mutations of contractile protein genes such as beta-cardiac myosin, myosin binding protein-C, and troponin-T. Unlike troponin-T, beta-myosin is a constituent of slow skeletal muscle and its mutations generally have a better prognosis. In order to investigate the usefulness of electromyography in detecting skeletal muscle involvement in HCM, 46 patients were examined using both conventional electromyography (EMG) and quantitative electromyography (QEMG) methods. The QEMG involved motor unit potential (MUP) analysis, turns/amplitude (TAA) analysis, and power spectrum analysis of the interference pattern. Using conventional EMG, myopathic findings were demonstrated in 13 patients (28%). Receiver operating characteristic (ROC) analysis of the results of a discriminant function extracted using QEMG values, identified correctly 10 out of 11 normal controls and all 9 myopathic control patients, and displayed a 15% presence of myopathy (7 patients) among the cardiomyopathy group. The duration of MUPs was the most sensitive among the quantitative parameters in differentiating normal from myopathic subjects. Since skeletal muscle involvement may be due to distinct gene mutations, normal and myopathic EMG findings may reflect HCM subpopulations with a different genetic substrate.
Authors:
N Karandreas; P Stathis; A Anastasakis; A Rigopoulos; P Piperos; A Theopistou; C Stefanadis; P Toutouzas
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Publication Detail:
Type:  Comparative Study; Journal Article    
Journal Detail:
Title:  Muscle & nerve     Volume:  23     ISSN:  0148-639X     ISO Abbreviation:  Muscle Nerve     Publication Date:  2000 Dec 
Date Detail:
Created Date:  2000-12-12     Completed Date:  2000-12-22     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  7803146     Medline TA:  Muscle Nerve     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  1856-61     Citation Subset:  IM    
Copyright Information:
Copyright 2000 John Wiley & Sons, Inc.
Affiliation:
EMG Laboratory, Department of Neurology, Aeginition Hospital, University of Athens, Vas. Sofias 72-74, 11528 Athens, Greece. nekaran@hotmail.com
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MeSH Terms
Descriptor/Qualifier:
Adolescent
Adult
Aged
Cardiomyopathy, Hypertrophic / physiopathology*
Electromyography / methods*
Evoked Potentials, Motor
False Positive Reactions
Humans
Male
Middle Aged
Muscle, Skeletal / physiopathology*
Muscular Diseases / complications,  diagnosis*,  physiopathology
Reproducibility of Results

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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