Document Detail

Electrolyte balance in gastrointestinal disease.
MedLine Citation:
PMID:  13260927     Owner:  NLM     Status:  MEDLINE    
Even small losses of gastrointestinal secretions when combined with reduced intake of electrolytes may seriously disturb electrolyte balance. Knowledge of the ionic composition of secretions lost is essential in planning therapy. Loss of gastric contents usually results in excessive loss of chloride; in achlorhydria this is not the case. Loss of sodium and potassium may be large in either case and is often underestimated. Small bowel obstruction results in a more balanced loss of electrolyte which may not affect acidbase balance greatly. In diarrhea loss of base predominates, and may result in a large potassium deficit. Steatorrhea due to nontropical sprue results in large fecal losses of sodium, potassium and chloride, in addition to the large calcium and phosphorus loss. In chronic peptic ulcer excessive ingestion of milk and absorbable alkalies may result in hypercalcemia, azotemia and alkalosis, without hypercalciuria. Since renal function is usually adequate in the milder gastrointestinal disturbances, electrolyte and fluid replacement should be started early, and can be guided by generally available laboratory tests, the carbon dioxide combining power and serum chloride levels, provided the predominate ionic loss is known and potassium deficiency remedied. If this is done, development of serious fluid and electrolyte deficits can usually be prevented.
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  California medicine     Volume:  83     ISSN:  0008-1264     ISO Abbreviation:  Calif Med     Publication Date:  1955 Nov 
Date Detail:
Created Date:  1956-12-01     Completed Date:  2003-05-01     Revised Date:  2009-11-03    
Medline Journal Info:
Nlm Unique ID:  0410260     Medline TA:  Calif Med     Country:  Not Available    
Other Details:
Languages:  eng     Pagination:  339-42     Citation Subset:  OM    
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MeSH Terms
Body Fluids*
Gastrointestinal Diseases / complications*

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine

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