Document Detail


Efficient lentiviral transduction of liver requires cell cycling in vivo.
MedLine Citation:
PMID:  10615126     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Human-immunodeficiency-virus (HIV)-based lentiviral vectors are a promising tool for in vivo gene therapy. Unlike Moloney-murine-leukaemia-based retroviruses (MLV), lentiviruses are believed to stably transduce quiescent (non-cycling) cells in various organs. No previous studies, however, have directly established the cell-cycle status of any transduced cell type at the time of vector administration in vivo. In vitro studies using wild-type HIV or HIV-based vectors have shown that, in some cases, cell-cycle activation is required for infection, even though cellular mitosis is not an absolute requirement for integration. Even if the block in reverse transcription is overcome in quiescent T cells, productive infection by HIV cannot be rescued in the absence of cell-cycle activation. The potential use of these vectors for gene therapy prompted our study, which establishes a cell-cycle requirement for efficient transduction of hepatocytes in vivo.
Authors:
F Park; K Ohashi; W Chiu; L Naldini; M A Kay
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Nature genetics     Volume:  24     ISSN:  1061-4036     ISO Abbreviation:  Nat. Genet.     Publication Date:  2000 Jan 
Date Detail:
Created Date:  2000-02-10     Completed Date:  2000-02-10     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  9216904     Medline TA:  Nat Genet     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  49-52     Citation Subset:  IM; X    
Affiliation:
Department of Pediatrics, Stanford University, Stanford, California, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Cycle / genetics*
Cell Line
Female
Genetic Vectors
Humans
Lentivirus / genetics*
Liver / cytology,  enzymology,  metabolism*
Mice
Mice, Inbred C57BL
Mice, SCID
Rats
Rats, Inbred F344
Transduction, Genetic*
Grant Support
ID/Acronym/Agency:
R01-HL53682/HL/NHLBI NIH HHS

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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