Document Detail


Effects of resistin expression on glucose metabolism and hepatic insulin resistance.
MedLine Citation:
PMID:  19184634     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
In order to observe the effect of increased serum resistin on glucose metabolism, insulin sensitivity, and hepatic insulin resistance (IR), mice were intravenously injected with recombinant adenovirus carrying the resistin gene (Adv-resistin-EGFP). Changes in hepatic glucose metabolism were observed using the Periodic Acid-Schiff method. Hepatic AMP-activated protein kinase (AMPK) activation was assessed by Western blot analysis, and glucose-6-phosphatase (G6Pase) and phosphoenolpyruvate carboxykinase (PEPCK) mRNA expression was determined using real-time RT-PCR. Although no effect on fasting blood glucose was detected, increased fasting insulin levels, decreased glucose tolerance and insulin sensitivity, and reduced hepatic glycogen levels and AMPK activation were seen in the Adv-resistin-EGFP mice. Finally, elevated G6Pase and PEPCK mRNA expression levels were detected upon overexpression of resistin. Resistin may inhibit hepatic AMPK activity, which results in elevated expression of gluconeogenic enzymes thereby affecting glucose metabolism and leading to decreased glycogen storage that contributes to the development of hepatic IR.
Authors:
Fang-Ping Li; Juan He; Zhi-Zhen Li; Zhao-Fan Luo; Li Yan; Yan Li
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2009-01-31
Journal Detail:
Title:  Endocrine     Volume:  35     ISSN:  1355-008X     ISO Abbreviation:  Endocrine     Publication Date:  2009 Apr 
Date Detail:
Created Date:  2009-03-24     Completed Date:  2009-06-18     Revised Date:  2010-06-24    
Medline Journal Info:
Nlm Unique ID:  9434444     Medline TA:  Endocrine     Country:  United States    
Other Details:
Languages:  eng     Pagination:  243-51     Citation Subset:  IM    
Affiliation:
Department of Endocrinology and Metabolism Department, The Second Affiliated Hospital, Sun Yat-sen University, No. 107 Yanjiang West Road, Guangzhou 510120, China. Amiy36@hotmail.com
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MeSH Terms
Descriptor/Qualifier:
AMP-Activated Protein Kinases / metabolism
Adenoviridae / genetics
Animals
Blood Glucose / analysis
Blotting, Western
Enzyme Activation
Fasting
Gene Expression*
Genetic Vectors
Glucose / metabolism*
Glucose Intolerance
Glucose-6-Phosphatase / genetics
Glycogen / analysis
Insulin / blood
Insulin Resistance / physiology*
Liver / chemistry,  drug effects*,  enzymology
Male
Mice
Mice, Inbred C57BL
Phosphoenolpyruvate Carboxykinase (GTP) / genetics
RNA, Messenger / analysis
Recombinant Proteins / genetics
Resistin / genetics*,  physiology
Reverse Transcriptase Polymerase Chain Reaction
Chemical
Reg. No./Substance:
0/Blood Glucose; 0/RNA, Messenger; 0/Recombinant Proteins; 0/Resistin; 11061-68-0/Insulin; 50-99-7/Glucose; 9005-79-2/Glycogen; EC 2.7.11.1/AMP-Activated Protein Kinases; EC 3.1.3.9/Glucose-6-Phosphatase; EC 4.1.1.32/Phosphoenolpyruvate Carboxykinase (GTP)

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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