Document Detail


Effects of omega-3 fatty acids on components of the transforming growth factor beta-1 pathway: implication for dietary modification and prevention in ovarian cancer.
MedLine Citation:
PMID:  19268879     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
OBJECTIVE: We previously demonstrated that omega-3 fatty acids (OM-3FAs) have definitive inhibitory effects on ovarian cancer cell lines. We sought to determine whether the inhibitory effects of OM-3FAs were mediated by the transforming growth factor (TGF)-beta1 signaling pathway. STUDY DESIGN: Ovarian cancer cell lines were grown at 37 degrees C in 5% CO(2) and treated with OM-3FAs, omega-6 fatty acids, and control at different concentrations for 24-72 hours. Enzyme-linked immunosorbent assay (ELISA) assay and Western blot analysis were used to measure TGF-beta1, phosphorylated mothers against decapentaplegic (Smad)-3 and p21 protein levels. RESULTS: An ELISA assay demonstrated that OM-3FA treatment increased TGF-beta1 in all 3 Hey cell lines (P < .05). In both SKOV-3 and OVCAR-3 cells, TGF-beta1 levels were not significantly increased. Western blots confirmed increases in TGF-beta1, Smad-3 and p21 protein levels in Hey and HeyC2 but not SKOV-3 and OVCAR-3 cells. CONCLUSION: OM-3FAs increased the level of TGF-beta1, Smad-3, and p21 protein in ovarian cancer cells known to be more sensitive to their inhibitory effect.
Authors:
Arun Sharma; Jennifer Belna; Joseph Espat; Gustavo Rodriguez; Vernon T Cannon; Jean A Hurteau
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Publication Detail:
Type:  Journal Article     Date:  2009-03-09
Journal Detail:
Title:  American journal of obstetrics and gynecology     Volume:  200     ISSN:  1097-6868     ISO Abbreviation:  Am. J. Obstet. Gynecol.     Publication Date:  2009 May 
Date Detail:
Created Date:  2009-04-20     Completed Date:  2009-05-18     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0370476     Medline TA:  Am J Obstet Gynecol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  516.e1-6     Citation Subset:  AIM; IM    
Affiliation:
Division of Gynecologic Oncology, Department of Obstetrics and Gynecology, University of Illinois Medical Center at Chicago, Chicago, IL, USA.
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MeSH Terms
Descriptor/Qualifier:
Benzamides
Cell Line, Tumor
Cyclin-Dependent Kinase Inhibitor p21 / metabolism
Fatty Acids, Omega-3 / pharmacology*
Fatty Acids, Omega-6 / pharmacology
Female
Humans
Ovarian Neoplasms / diet therapy,  metabolism*,  prevention & control*
Phosphorylation / drug effects
Signal Transduction / drug effects*
Smad3 Protein / metabolism
Transforming Growth Factor beta1 / metabolism*
Tumor Suppressor Protein p53 / metabolism
Chemical
Reg. No./Substance:
0/Benzamides; 0/CDKN1A protein, human; 0/Cyclin-Dependent Kinase Inhibitor p21; 0/Fatty Acids, Omega-3; 0/Fatty Acids, Omega-6; 0/SMAD3 protein, human; 0/Smad3 Protein; 0/Transforming Growth Factor beta1; 0/Tumor Suppressor Protein p53; 82558-50-7/isoxaben

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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