Document Detail


Effects of insulin and insulin-like growth factor I on growth of human leukemia cells in serum-free and protein-free medium.
MedLine Citation:
PMID:  3291986     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Human myeloid leukemia cells (HL60) and malignant lymphocytes (Namalwa) were grown in protein-free, Fe-supplemented media and used to study growth responses to insulin and insulin-like growth factor 1 (IGF-I). HL60 cells previously grown in serum-free medium containing microgram quantities of insulin showed an 18-fold reduction in cumulative cell production when grown without insulin. However, the same cells showed reduced or absent growth stimulation with 1 to 100 ng/mL insulin or IGF-I for at least four days following insulin deprivation, indicating that culture conditions modified insulin and IGF-I responses. When the same cells were grown in Fe-supplemented, protein-free medium (RPMI-Fe), insulin and IGF-I caused dose-dependent stimulation of HL60 cell growth with half-maximal stimulation at nanogram concentrations. Namalwa cells grown in protein-free medium showed no response to either hormone. Radioligand binding showed the presence of insulin and IGF-I receptors on both HL60 and Namalwa cells grown in RPMI-Fe. HL60 cells grown in fetal bovine serum had higher, and cells grown with microgram quantities of insulin dramatically reduced, insulin binding. Competitive binding studies and cultures with anti-IGF-I receptor antibody showed insulin and IGF-I stimulated growth through their respective specific receptors. Both insulin and IGF-I stimulate growth of some cultured human leukemia cells, but the presence of insulin or IGF-I receptors alone does not predict growth responses. Culture conditions affect both cellular responses and ligand binding by these hormones and must be closely controlled to study growth responses.
Authors:
J Sinclair; D McClain; R Taetle
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Blood     Volume:  72     ISSN:  0006-4971     ISO Abbreviation:  Blood     Publication Date:  1988 Jul 
Date Detail:
Created Date:  1988-08-19     Completed Date:  1988-08-19     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  7603509     Medline TA:  Blood     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  66-72     Citation Subset:  AIM; IM    
Affiliation:
Department of Medicine, University of California, San Diego 92103.
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MeSH Terms
Descriptor/Qualifier:
Antibodies, Monoclonal / physiology
Binding, Competitive
Cell Division / drug effects*
Cell Line
Culture Media / analysis*
Dose-Response Relationship, Drug
Fetal Blood
Humans
Insulin / metabolism,  pharmacology*
Insulin-Like Growth Factor I / metabolism,  pharmacology*
Leukemia / metabolism,  pathology*
Proteins
Receptor, Insulin / analysis,  drug effects,  immunology
Somatomedins / pharmacology*
Time Factors
Tumor Cells, Cultured / metabolism,  pathology*
Grant Support
ID/Acronym/Agency:
CA09290/CA/NCI NIH HHS; CA23100/CA/NCI NIH HHS; CA32094/CA/NCI NIH HHS
Chemical
Reg. No./Substance:
0/Antibodies, Monoclonal; 0/Culture Media; 0/Proteins; 0/Somatomedins; 11061-68-0/Insulin; 67763-96-6/Insulin-Like Growth Factor I; EC 2.7.10.1/Receptor, Insulin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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