Document Detail


Effects of in vitro amiodarone exposure on alveolar macrophage inflammatory mediator production.
MedLine Citation:
PMID:  1333729     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Administration of amiodarone, although often lifesaving, is associated with pulmonary side effects. Patients with amiodarone pulmonary toxicity can present with either a chronic disorder that suggests pulmonary fibrosis or a more acute process. Mechanisms of acute pulmonary injury resulting from amiodarone are unclear. Previous studies have demonstrated that the drug is preferentially concentrated in alveolar macrophages. In the present study, the authors examined whether in vitro exposure to amiodarone resulted in alteration of rat alveolar macrophage superoxide, leukotriene B4, or fibronectin release. In addition, the authors assessed whether macrophages were ultrastructurally altered by in vitro amiodarone exposure. Twenty four hour exposure to therapeutic tissue concentrations of amiodarone resulted in enhancement of phorbol myristate acetate-stimulated macrophage superoxide release. In addition, 48 hours exposure to amiodarone caused a dose-dependent inhibition of spontaneous fibronectin release by macrophages. Macrophages exposed to 48 hours of 10 micrograms/ml amiodarone were ultrastructurally abnormal, containing lamellar inclusions and demonstrating a large degree of vacuolization. The authors concluded that alveolar macrophages are very sensitive to therapeutic tissue concentrations of amiodarone. Alteration of macrophage mediator release by amiodarone may be one mechanism for lung damage induced by the drug.
Authors:
R J Zitnik; J A Cooper; J A Rankin; J Sussman
Related Documents :
6288939 - Pretreatment with phorbol myristate acetate inhibits macrophage activity against intrac...
15591219 - Pitavastatin inhibits remnant lipoprotein-induced macrophage foam cell formation throug...
18454339 - Monocyte phagocytosis of viable staphylococcus aureus is impaired by barbiturates, but ...
22569899 - Pharmacodynamic effects of cangrelor and clopidogrel: the platelet function substudy fr...
2166359 - Effects of an inhaled corticosteroid, budesonide, on alveolar macrophage function in sm...
18974159 - Inhibitory effect of n-palmitoylphosphatidylethanolamine on macrophage phagocytosis thr...
209059 - Selective uptake of the synthetic amino terminal fragment of bovine parathyroid hormone...
1535649 - Enhanced platelet function in acute myocardial infarction is attenuated by streptokinas...
10552949 - Factor xi messenger rna in human platelets.
Publication Detail:
Type:  In Vitro; Journal Article; Research Support, U.S. Gov't, Non-P.H.S.    
Journal Detail:
Title:  The American journal of the medical sciences     Volume:  304     ISSN:  0002-9629     ISO Abbreviation:  Am. J. Med. Sci.     Publication Date:  1992 Dec 
Date Detail:
Created Date:  1993-01-06     Completed Date:  1993-01-06     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0370506     Medline TA:  Am J Med Sci     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  352-6     Citation Subset:  AIM; IM    
Affiliation:
Pulmonary Section, Yale University School of Medicine, New Haven, Connecticut.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Amiodarone / adverse effects*
Animals
Cell Adhesion / drug effects
Fibronectins / metabolism
Inflammation / pathology
Leukotriene B4 / metabolism
Macrophages, Alveolar / drug effects*,  metabolism,  ultrastructure
Male
Microscopy, Electron
Rats
Superoxides / metabolism
Chemical
Reg. No./Substance:
0/Fibronectins; 11062-77-4/Superoxides; 1951-25-3/Amiodarone; 71160-24-2/Leukotriene B4

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Orthodontic microabrasive reproximation.
Next Document:  Melanotic neuroectodermal tumor of infancy. A case report of paratesticular primary with lymph node ...