Document Detail


Effects of glucosinolate-rich broccoli sprouts on urinary levels of aflatoxin-DNA adducts and phenanthrene tetraols in a randomized clinical trial in He Zuo township, Qidong, People's Republic of China.
MedLine Citation:
PMID:  16284385     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Residents of Qidong, People's Republic of China, are at high risk for development of hepatocellular carcinoma, in part due to consumption of aflatoxin-contaminated foods, and are exposed to high levels of phenanthrene, a sentinel of hydrocarbon air toxics. Cruciferous vegetables, such as broccoli, contain anticarcinogens. Glucoraphanin, the principal glucosinolate in broccoli sprouts, can be hydrolyzed by gut microflora to sulforaphane, a potent inducer of carcinogen detoxication enzymes. In a randomized, placebo-controlled chemoprevention trial, we tested whether drinking hot water infusions of 3-day-old broccoli sprouts, containing defined concentrations of glucosinolates, could alter the disposition of aflatoxin and phenanthrene. Two hundred healthy adults drank infusions containing either 400 or < 3 micromol glucoraphanin nightly for 2 weeks. Adherence to the study protocol was outstanding; no problems with safety or tolerance were noted. Urinary levels of aflatoxin-N(7)-guanine were not different between the two intervention arms (P = 0.68). However, measurement of urinary levels of dithiocarbamates (sulforaphane metabolites) indicated striking interindividual differences in bioavailability. An inverse association was observed for excretion of dithiocarbamates and aflatoxin-DNA adducts (P = 0.002; R = 0.31) in individuals receiving broccoli sprout glucosinolates. Moreover, trans, anti-phenanthrene tetraol, a metabolite of the combustion product phenanthrene, was detected in urine of all participants and showed a robust inverse association with dithiocarbamate levels (P = 0.0001; R = 0.39), although again no overall difference between intervention arms was observed (P = 0.29). Understanding factors influencing glucosinolate hydrolysis and bioavailability will be required for optimal use of broccoli sprouts in human interventions.
Authors:
Thomas W Kensler; Jian-Guo Chen; Patricia A Egner; Jed W Fahey; Lisa P Jacobson; Katherine K Stephenson; Lingxiang Ye; Jamie L Coady; Jin-Bing Wang; Yan Wu; Yan Sun; Qi-Nan Zhang; Bao-Chu Zhang; Yuan-Rong Zhu; Geng-Sun Qian; Stephen G Carmella; Stephen S Hecht; Lorie Benning; Stephen J Gange; John D Groopman; Paul Talalay
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Publication Detail:
Type:  Journal Article; Randomized Controlled Trial; Research Support, N.I.H., Extramural    
Journal Detail:
Title:  Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology     Volume:  14     ISSN:  1055-9965     ISO Abbreviation:  Cancer Epidemiol. Biomarkers Prev.     Publication Date:  2005 Nov 
Date Detail:
Created Date:  2005-11-14     Completed Date:  2006-01-10     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  9200608     Medline TA:  Cancer Epidemiol Biomarkers Prev     Country:  United States    
Other Details:
Languages:  eng     Pagination:  2605-13     Citation Subset:  IM    
Affiliation:
Department of Environmental Health Sciences, Bloomberg School of Public Health, Johns Hopkins University, Room E7541, 615 North Wolfe Street, Baltimore, MD 21205, USA. tkensler@jhsph.edu
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MeSH Terms
Descriptor/Qualifier:
Adult
Aflatoxins / metabolism,  urine*
Aged
Anticarcinogenic Agents / pharmacology*
Beverages
Biological Availability
Brassica / chemistry*
Carcinoma, Hepatocellular / etiology,  prevention & control
DNA Adducts / urine*
Female
Glucosinolates / pharmacology*
Humans
Hydrolysis
Liver Neoplasms / etiology,  prevention & control
Male
Middle Aged
Phenanthrenes / urine*
Placebos
Grant Support
ID/Acronym/Agency:
ES 03819/ES/NIEHS NIH HHS; P01 ES 06052/ES/NIEHS NIH HHS; R01 CA 92025/CA/NCI NIH HHS; R01 CA 93780/CA/NCI NIH HHS; R21 CA 098485/CA/NCI NIH HHS
Chemical
Reg. No./Substance:
0/Aflatoxins; 0/Anticarcinogenic Agents; 0/DNA Adducts; 0/Glucosinolates; 0/Phenanthrenes; 0/Placebos

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