Document Detail


Effects of enadoline on the development of pentylenetetrazol kindling, learning performance, and hippocampal morphology.
MedLine Citation:
PMID:  10095026     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Opioids are involved in the development of epileptic seizures. Recently, interest has been focused on the role of the kappa-opioid receptor agonists as novel approaches to the treatment of epilepsy. In the present study we investigated the effects of the kappa-opioid receptor agonist enadoline (Ena) on pentylenetetrazol (PTZ) induced seizures, PTZ kindling, shuttle-box performance and hippocampal neuromorphology. Ena injected i.c.v. in doses of 1 and 10 nmol did not affect acute PTZ seizures. In the course of PTZ kindling development, co-treatment (1 nmol) with the kappa-opioid receptor agonist suppressed seizure strength. Eight days after kindling completion the animals received a challenge dose of PTZ. In reaction to challenge, kindled animals which were pretreated with Ena reached significantly lower seizure scores. Kindling resulted in diminished shuttle-box performance. Learning performance in kindled animals pretreated with Ena was not normalised. Kindling resulted in increased glutamate binding. Interestingly, in comparison with the saline/saline group, neither in the Ena/saline nor in the Ena/PTZ treated groups changes in glutamate binding were found. That means that Ena prevented the increase in glutamate binding in the kindled group. In kindled animals significant cell loss in CA1 of the dorsal hippocampus was found and this was efficaciously counteracted by Ena. However, Ena alone did induce similar cell loss compared to kindled animals. It is hypothesised that the effects of enadoline are mainly due to interferences with glutamatergic systems.
Authors:
A Becker; H Braun; H Schröder; G Grecksch; V Höllt
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Brain research     Volume:  823     ISSN:  0006-8993     ISO Abbreviation:  Brain Res.     Publication Date:  1999 Mar 
Date Detail:
Created Date:  1999-05-07     Completed Date:  1999-05-07     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0045503     Medline TA:  Brain Res     Country:  NETHERLANDS    
Other Details:
Languages:  eng     Pagination:  191-7     Citation Subset:  IM    
Copyright Information:
Copyright 1999 Elsevier Science B.V.
Affiliation:
O.-v.-Guericke University, Faculty of Medicine, Institute of Pharmacology and Toxicology, Leipzigerstr. 44, D-39120, Magdeburg, Germany. axel.becker@medizin.uni-magdeburg.de
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MeSH Terms
Descriptor/Qualifier:
Animals
Benzofurans / pharmacology*
Convulsants / pharmacology*
Glutamic Acid / metabolism
Hippocampus / drug effects*,  metabolism,  pathology*
Kindling, Neurologic / drug effects*
Learning / drug effects*
Male
Pentylenetetrazole / pharmacology*
Pyrrolidines / pharmacology*
Rats
Rats, Wistar
Seizures / chemically induced,  physiopathology
Chemical
Reg. No./Substance:
0/Benzofurans; 0/Convulsants; 0/Pyrrolidines; 107431-28-7/enadoline; 54-95-5/Pentylenetetrazole; 56-86-0/Glutamic Acid

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