Document Detail


Effects of components of meals (carbohydrate, fat, protein) in causing postprandial exertional angina pectoris.
MedLine Citation:
PMID:  9165167     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
We have shown that unlike fat, protein, xylose, or water, the carbohydrate component of the meal accelerates myocardial ischemia, reduces exercise capacity, and is associated with a more rapid increase in the determinants of myocardial oxygen consumption than exercise in the fasting state. Our results suggest a role for a larger increase in sympathetic nervous activity and/or release of vasoactive gastrointestinal peptides after carbohydrate, but not fat or protein, meals in postprandial angina.
Authors:
R R Baliga; L Burden; M K Sidhu; M W Rampling; J S Kooner
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  The American journal of cardiology     Volume:  79     ISSN:  0002-9149     ISO Abbreviation:  Am. J. Cardiol.     Publication Date:  1997 May 
Date Detail:
Created Date:  1997-06-19     Completed Date:  1997-06-19     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  0207277     Medline TA:  Am J Cardiol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  1397-400     Citation Subset:  AIM; IM    
Affiliation:
Department of Medicine, Royal Postgraduate Medical School, Hammersmith Hospital, London, United Kingdom.
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MeSH Terms
Descriptor/Qualifier:
Angina Pectoris / blood,  etiology*,  physiopathology
Catecholamines / blood
Dietary Carbohydrates / administration & dosage*
Dietary Fats / administration & dosage*
Dietary Proteins / administration & dosage*
Electrocardiography
Erythrocyte Aggregation
Exercise Test
Hemodynamics
Hemostasis
Humans
Insulin / blood
Male
Physical Exertion*
Postprandial Period
Xylose / administration & dosage
Chemical
Reg. No./Substance:
0/Catecholamines; 0/Dietary Carbohydrates; 0/Dietary Fats; 0/Dietary Proteins; 0/Xylose; 11061-68-0/Insulin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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