Document Detail


Effects of carvedilol on oxidative stress and chronotropic response to exercise in patients with chronic heart failure.
MedLine Citation:
PMID:  16227141     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: Our previous studies suggest that the increase in heart rate from rest to peak exercise is reduced in patients with chronic heart failure (CHF) and this is associated with increased oxidative stress, as determined by malondialdehyde (MDA) plasma levels.
AIM: To investigate the effects of carvedilol on the heart rate response to exercise and oxidative stress in patients with CHF.
METHODS AND RESULTS: Thirty stable NYHA classes II-III CHF patients received carvedilol therapy for 6 months, at a mean maintenance dose of 25 mg (range 6.25-50 mg/day). After treatment, the patients showed a significant improvement in their functional NYHA class (p=0.013), increased left ventricular ejection fraction (LVEF) (24+/-1.4% to 31+/-2.3%, p=0.003) and 6-min walk distance (499+/-18 to 534+/-18 m, p=0.03), without changes in the peak VO2. At baseline, norepinephrine (NE) plasma levels increased with exercise (510+/-51 to 2513+/-230 pg/mL, p<0.001), and these levels were not affected by carvedilol. Chronotropic responsiveness index (increase in heart rate divided by the increase in NE from rest to peak exercise) was not changed by carvedilol (0.049+/-0.001 to 0.042+/-0.001, p=0.6). MDA levels of CHF patients decreased after treatment with carvedilol (2.4+/-0.2 to 1.1+/-0.2 microM, p<0.001), without changes in antioxidant enzyme activities.
CONCLUSIONS: Carvedilol treatment in patients with CHF results in reduced oxidative stress without restoration of the chronotropic responsiveness index.
Authors:
Pablo Castro; José Luis Vukasovic; Mario Chiong; Guillermo Díaz-Araya; Hernán Alcaino; Miguel Copaja; Rodrigo Valenzuela; Douglas Greig; Osvaldo Pérez; Ramón Corbalan; Sergio Lavandero
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  European journal of heart failure     Volume:  7     ISSN:  1388-9842     ISO Abbreviation:  Eur. J. Heart Fail.     Publication Date:  2005 Oct 
Date Detail:
Created Date:  2005-10-17     Completed Date:  2006-01-23     Revised Date:  2011-06-08    
Medline Journal Info:
Nlm Unique ID:  100887595     Medline TA:  Eur J Heart Fail     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  1033-9     Citation Subset:  IM    
Affiliation:
Cardiovascular Diseases, Universidad Católica de Chile, Santiago, Chile. pcastro@med.puc.cl
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MeSH Terms
Descriptor/Qualifier:
Adrenergic beta-Antagonists / therapeutic use*
Aged
Carbazoles / therapeutic use*
Dose-Response Relationship, Drug
Drug Administration Schedule
Female
Follow-Up Studies
Glutathione Peroxidase / analysis,  metabolism
Heart Failure / drug therapy*,  enzymology*,  radionuclide imaging
Heart Function Tests
Heart Rate / drug effects*
Humans
Linear Models
Male
Malondialdehyde / analysis,  metabolism
Middle Aged
Norepinephrine / metabolism
Oxidative Stress / drug effects*,  physiology
Probability
Propanolamines / therapeutic use*
Prospective Studies
Radionuclide Ventriculography / drug effects
Severity of Illness Index
Stroke Volume / drug effects
Superoxide Dismutase / analysis,  metabolism
Chemical
Reg. No./Substance:
0/Adrenergic beta-Antagonists; 0/Carbazoles; 0/Propanolamines; 51-41-2/Norepinephrine; 542-78-9/Malondialdehyde; 72956-09-3/carvedilol; EC 1.11.1.9/Glutathione Peroxidase; EC 1.15.1.1/Superoxide Dismutase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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