Document Detail


Effects of cAMP on intercellular coupling and osteoblast differentiation.
MedLine Citation:
PMID:  11302734     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Bone-forming cells are organized in a multicellular network interconnected by gap junctions. Direct intercellular communication via gap junctions is an important component of bone homeostasis, coordinating cellular responses to external signals and promoting osteoblast differentiation. The cAMP pathway, a major intercellular signal transduction mechanism, regulates osteoblastic function and metabolism. We investigated the effects of this second messenger on junctional communication and on the expression of differentiation markers in human HOBIT osteoblastic cells. Increased levels of cAMP induce posttranslational modifications (i.e., phosphorylations) of connexin43 and enhancement of gap junction assembly, resulting in an increased junctional permeance to Lucifer yellow and to a positive modulation of intercellular Ca(2+) waves. Increased intercellular communication, however, was accompanied by a parallel decrease of alkaline phosphatase activity and by an increase of osteocalcin expression. cAMP-dependent stimulation of cell-to-cell coupling induces a complex modulation of bone differentiation markers.
Authors:
M Romanello; L Moro; D Pirulli; S Crovella; P D'Andrea
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Biochemical and biophysical research communications     Volume:  282     ISSN:  0006-291X     ISO Abbreviation:  Biochem. Biophys. Res. Commun.     Publication Date:  2001 Apr 
Date Detail:
Created Date:  2001-04-16     Completed Date:  2001-05-17     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0372516     Medline TA:  Biochem Biophys Res Commun     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1138-44     Citation Subset:  IM    
Copyright Information:
Copyright 2001 Academic Press.
Affiliation:
Dipartimento di Biochimica, Biofisica e Chimica delle Macromolecole, Università di Trieste, via Licio Giorgieri 1, Trieste, I-34127, Italy.
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MeSH Terms
Descriptor/Qualifier:
Alkaline Phosphatase / antagonists & inhibitors,  metabolism
Antigens, Differentiation / metabolism
Calcium Signaling / drug effects
Cell Communication / drug effects,  physiology*
Cell Differentiation / drug effects,  physiology*
Cell Line
Cell Membrane Permeability / drug effects
Connexin 43 / metabolism
Cyclic AMP / metabolism*,  pharmacology
Forskolin / pharmacology
Gap Junctions / drug effects
Humans
Osteoblasts / cytology,  drug effects,  metabolism*
Osteocalcin / genetics,  metabolism
Phosphodiesterase Inhibitors / pharmacology
Phosphorylation / drug effects
RNA, Messenger / metabolism
Signal Transduction / drug effects,  physiology
Chemical
Reg. No./Substance:
0/Antigens, Differentiation; 0/Connexin 43; 0/Phosphodiesterase Inhibitors; 0/RNA, Messenger; 104982-03-8/Osteocalcin; 60-92-4/Cyclic AMP; 66428-89-5/Forskolin; EC 3.1.3.1/Alkaline Phosphatase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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