Document Detail


Effects of asbestos fibers on alveolar macrophage-mediated lymphocyte cytostasis.
MedLine Citation:
PMID:  3709494     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Prolonged asbestos inhalation results in pulmonary inflammation and fibrosis. Since alveolar macrophages are active in regulation of immune responses in lung and appear to be involved in the pathogenesis of asbestosis, we evaluated the effects of asbestos exposure on the ability of these cells to regulate lymphocyte function. Alveolar macrophages obtained by lung lavage from BALB/C mice were treated in vitro with either UICC amosite or chrysotile asbestos and the effects on lymphocyte cytostasis compared with those of macrophages incubated with latex beads or zymosan. Macrophages (10%) incubated either alone or with latex beads for 48 hr effectively inhibited lymphocyte mitogenesis. However, alveolar macrophages incubated with either amosite or chrysotile asbestos did not demonstrate intact cytostatic activity. Decreased viability of chrysotile asbestos-treated macrophages correlated with loss of cytostatic effects, but alveolar macrophages exposed to amosite remained viable. We conclude, therefore, that exposure of alveolar macrophages to asbestos can result in loss of their ability to down-regulate lymphocyte proliferation, a finding which may be important in the pathogenesis of asbestos-related disease.
Authors:
B E Bozelka; P Sestini; Y Hammad; J E Salvaggio
Related Documents :
3862614 - Mechanisms of tobacco smoke toxicity on pulmonary macrophage cells.
570554 - Proteoglycans, proteases, chemotaxis, and aggregation of inflammatory, cells.
11122454 - A critical role for alveolar macrophages in elicitation of pulmonary immune fibrosis.
15516494 - Ccr2-positive monocytes recruited to inflamed lungs downregulate local ccl2 chemokine l...
19854464 - Enhanced transformation of triclosan by laccase in the presence of redox mediators.
16817774 - Differential expression of ovine innate immune genes by preterm and neonatal lung epith...
Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Environmental research     Volume:  40     ISSN:  0013-9351     ISO Abbreviation:  Environ. Res.     Publication Date:  1986 Jun 
Date Detail:
Created Date:  1986-06-30     Completed Date:  1986-06-30     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0147621     Medline TA:  Environ Res     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  172-80     Citation Subset:  IM    
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Asbestos / immunology*,  pharmacology
Asbestosis / etiology,  immunology*
Cell Survival
Cells, Cultured
Female
Lymphocyte Activation / drug effects
Mice
Mice, Inbred BALB C
Pulmonary Alveoli / immunology*
Spleen / immunology
Grant Support
ID/Acronym/Agency:
HL-15092-13/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
1332-21-4/Asbestos

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Ultrastructural changes in gills of Sarotherodon mossambicus treated with chicken manure.
Next Document:  Hypertension, age, and smoking related to genotoxic sensitivity in human lymphocytes: a population s...