Document Detail


Effects of non-HLA gene polymorphisms on development of islet autoimmunity and type 1 diabetes in a population with high-risk HLA-DR,DQ genotypes.
MedLine Citation:
PMID:  22315323     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
We assessed the effects of non-HLA gene polymorphisms on the risk of islet autoimmunity (IA) and progression to type 1 diabetes in the Diabetes Autoimmunity Study in the Young. A total of 1,743 non-Hispanic, white children were included: 861 first-degree relatives and 882 general population children identified as having high-risk HLA-DR/DQ genotypes for type 1 diabetes. Of those, 109 developed IA and 61 progressed to diabetes. Study participants were genotyped for 20 non-HLA polymorphisms, previously confirmed as type 1 diabetes susceptibility loci. PTPN22 and UBASH3A predicted both IA and diabetes in regression models controlling for family history of type 1 diabetes and presence of HLA-DR3/4-DQB1*0302 genotype. In addition, PTPN2 predicted IA whereas INS predicted type 1 diabetes. The final multivariate regression models for both IA and type 1 diabetes included PTPN22, UBASH3A, and INS, in addition to family history of type 1 diabetes and HLA-DR3/4. In general population children, the most frequent combinations including these five significant predictors conferred hazard ratio of up to 13 for IA and >40 for type 1 diabetes. Non-HLA susceptibility alleles may help estimate risk for development of type 1 diabetes in the general population. These findings require replication in different populations.
Authors:
Andrea K Steck; Randall Wong; Brandie Wagner; Kelly Johnson; Edwin Liu; Jihane Romanos; Cisca Wijmenga; Jill M Norris; George S Eisenbarth; Marian J Rewers
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2012-02-07
Journal Detail:
Title:  Diabetes     Volume:  61     ISSN:  1939-327X     ISO Abbreviation:  Diabetes     Publication Date:  2012 Mar 
Date Detail:
Created Date:  2012-02-22     Completed Date:  2012-04-10     Revised Date:  2012-04-12    
Medline Journal Info:
Nlm Unique ID:  0372763     Medline TA:  Diabetes     Country:  United States    
Other Details:
Languages:  eng     Pagination:  753-8     Citation Subset:  AIM; IM    
Affiliation:
Barbara Davis Center for Childhood Diabetes, University of Colorado Denver, Aurora, Colorado, USA. andrea.steck@ucdenver.edu
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MeSH Terms
Descriptor/Qualifier:
Adaptor Proteins, Signal Transducing / genetics
Autoimmunity*
Diabetes Mellitus, Type 1 / genetics*
Genotype
HLA-DQ Antigens / genetics*
HLA-DR Antigens / genetics*
Humans
Islets of Langerhans / immunology*
Polymorphism, Single Nucleotide*
Protein Tyrosine Phosphatase, Non-Receptor Type 22 / genetics
Risk Factors
Grant Support
ID/Acronym/Agency:
AI-050864/AI/NIAID NIH HHS; DK-050979/DK/NIDDK NIH HHS; DK-320083/DK/NIDDK NIH HHS; DK-57516/DK/NIDDK NIH HHS; N01-AI-15416/AI/NIAID NIH HHS; R01 DK032493-29/DK/NIDDK NIH HHS; R37-DK-32493/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Adaptor Proteins, Signal Transducing; 0/HLA-DQ Antigens; 0/HLA-DR Antigens; 0/UBASH3A protein, human; EC 3.1.3.48/PTPN22 protein, human; EC 3.1.3.48/Protein Tyrosine Phosphatase, Non-Receptor Type 22

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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