Document Detail


Effects of NG-nitro-L-arginine methyl ester on regional haemodynamic responses to MgSO4 in conscious rats.
MedLine Citation:
PMID:  8012714     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
1. We assessed regional haemodynamic responses to the vasodilator, MgSO4, in the absence and presence of the nitric oxide synthase inhibitor, NG-nitro-L-arginine methyl ester (L-NAME), in conscious chronically instrumented Long Evans rats (n = 9). 2. MgSO4 (loading dose 220 mumol kg-1 min-1 for 7 min, maintenance dose 56 mumol kg-1 min-1 for 7 min), alone, caused slight bradycardia and hypotension accompanied by reductions in renal and mesenteric flows, but a marked hyperaemic vasodilatation in the hindquarters (flow, delta 54 +/- 6%, vascular conductance, delta 77 +/- 5%). 3. L-NAME (183 nmol kg-1 min-1) caused hypertension (29 +/- 2 mmHg) accompanied by bradycardia (-51 +/- 6 beats min-1) and reductions in flow and vascular conductance in the renal (-18 +/- 4% and -35 +/- 3%, respectively), mesenteric (-35 +/- 3% and -49 +/- 3%, respectively), and hindquarters (-26 +/- 3% and -42 +/- 3%, respectively) vascular beds. In the presence of L-NAME, the hypotensive and bradycardic effects of MgSO4 were still apparent, but its hindquarters hyperaemic vasodilator effect was significantly attenuated. 4. In order to determine if the inhibitory action of L-NAME on the hindquarters hyperaemic vasodilator action of MgSO4 was a non-specific effect, due to the change in baseline conditions caused by L-NAME, we also examined responses to MgSO4 in the presence of endothelin-1 (12.5 pmol kg-1 min-1) or angiotensin II (50 pmol kg-1 min-1). In the presence of either peptide, the overall effects of MgSO4 on hindquarters flow and vascular conductance were unchanged. 5. In a separate experiment (n = 8) we determined that the inhibitory effect of L-NAME on the hyperaemic vasodilator response to MgSO4 was prevented by L-arginine, and also demonstrated that the Beta2-adrenoceptor antagonist, ICI 118551, caused significant inhibition of the hindquarters haemodynamic effects of MgSO4.6. We conclude that the hindquarters haemodynamic effects of MgSO4 in conscious rats involve a substantial L-NAME-sensitive component which depends on activation of Beta2-adrenoceptors, probably asa consequence of adrenal medullary adrenaline release.
Authors:
P A Kemp; S M Gardiner; J E March; T Bennett; P C Rubin
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  British journal of pharmacology     Volume:  111     ISSN:  0007-1188     ISO Abbreviation:  Br. J. Pharmacol.     Publication Date:  1994 Jan 
Date Detail:
Created Date:  1994-07-25     Completed Date:  1994-07-25     Revised Date:  2009-11-18    
Medline Journal Info:
Nlm Unique ID:  7502536     Medline TA:  Br J Pharmacol     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  325-31     Citation Subset:  IM    
Affiliation:
Department of Physiology & Pharmacology, University of Nottingham Medical School, Queen's Medical Centre.
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MeSH Terms
Descriptor/Qualifier:
Angiotensin II / pharmacology
Animals
Arginine / administration & dosage,  analogs & derivatives*,  pharmacology
Blood Pressure / drug effects
Drug Interactions
Drug Synergism
Endothelins / pharmacology
Heart Rate / drug effects
Hemodynamics / drug effects*
Magnesium Sulfate / pharmacology*
Male
Mesenteric Arteries / drug effects
NG-Nitroarginine Methyl Ester
Nitric Oxide / antagonists & inhibitors*
Propanolamines / pharmacology
Rats
Receptors, Adrenergic, beta / drug effects*
Renal Artery / drug effects
Vascular Resistance / drug effects
Vasodilation / drug effects
Chemical
Reg. No./Substance:
0/Endothelins; 0/Propanolamines; 0/Receptors, Adrenergic, beta; 10102-43-9/Nitric Oxide; 11128-99-7/Angiotensin II; 50903-99-6/NG-Nitroarginine Methyl Ester; 72795-19-8/ICI 118551; 74-79-3/Arginine; 7487-88-9/Magnesium Sulfate
Comments/Corrections

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