Document Detail


Effects of K(+) channel inhibitors on relaxation induced by flufenamic and tolfenamic acids in guinea-pig trachea.
MedLine Citation:
PMID:  10585531     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The effects of different K(+) channel inhibitors on flufenamic- and tolfenamic-acids-induced relaxation were studied in prostaglandin F(2alpha) (1 microM) precontracted guinea-pig trachea. Flufenamic and tolfenamic acids (each 0.1-33 microM) and lemakalim (0.01-33 microM), but not indomethacin (0.1-33 microM), caused relaxation. Iberiotoxin (33 and 100 nM) inhibited flufenamic- and tolfenamic-acids-, but not lemakalim-, induced relaxation. Iberiotoxin (100 nM) inhibited nifedipine (10 nM-10 microM)-induced relaxation. Nifedipine (0.1 microM) inhibited the blockade of fenamate-induced relaxation by iberiotoxin. Apamin (0.1 and 1 microM) did not affect flufenamic- and tolfenamic-acids- and lemakalim-induced relaxation. Glibenclamide (10 and 33 microM) inhibited lemakalim-, but not flufenamic- and tolfenamic-acids-, induced relaxation. 4-Aminopyridine (0.5 and 2 mM) inhibited flufenamic- and tolfenamic- acids- and lemakalim-induced relaxation. Flufenamic- and tolfenamic-acids-induced relaxation is likely to be activation of Ca(2+)-activated K(+) channels and differs from that of lemakalim.
Authors:
L Li; K Vaali; H Vapaatalo; H Kankaanranta
Publication Detail:
Type:  In Vitro; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  European journal of pharmacology     Volume:  383     ISSN:  0014-2999     ISO Abbreviation:  Eur. J. Pharmacol.     Publication Date:  1999 Oct 
Date Detail:
Created Date:  2000-02-03     Completed Date:  2000-02-03     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  1254354     Medline TA:  Eur J Pharmacol     Country:  NETHERLANDS    
Other Details:
Languages:  eng     Pagination:  169-76     Citation Subset:  IM    
Affiliation:
Institute of Biomedicine, Department of Pharmacology and Toxicology, P.O. Box 8, University of Helsinki, Helsinki, Finland. liangli@ruby.iupui.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Anthranilic Acids / antagonists & inhibitors*,  pharmacology
Anti-Inflammatory Agents, Non-Steroidal / antagonists & inhibitors*,  pharmacology
Calcium Channel Blockers / pharmacology
Calcium Channels, L-Type / drug effects
Cromakalim / pharmacology
Dinoprost / pharmacology
Female
Flufenamic Acid / antagonists & inhibitors*,  pharmacology
Guinea Pigs
Indomethacin / pharmacology
Male
Muscle Relaxation / drug effects
Nifedipine / pharmacology
Potassium Channel Blockers*
Potassium Channels / agonists
Trachea / drug effects*
Chemical
Reg. No./Substance:
0/Anthranilic Acids; 0/Anti-Inflammatory Agents, Non-Steroidal; 0/Calcium Channel Blockers; 0/Calcium Channels, L-Type; 0/Potassium Channel Blockers; 0/Potassium Channels; 13710-19-5/tolfenamic acid; 21829-25-4/Nifedipine; 53-86-1/Indomethacin; 530-78-9/Flufenamic Acid; 551-11-1/Dinoprost; 94470-67-4/Cromakalim

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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