Document Detail


Effects of IFN-gamma and Stat1 on gene expression, growth, and survival in non-small cell lung cancer cells.
MedLine Citation:
PMID:  17348819     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Stat transcription factors are activated by cytokines and can activate pathways important in oncogenesis. Although previous studies have identified an oncogenic role of Stat3 in lung cancer cells, the role of Stat1 is unclear. Using a mutant of Stat1 with constitutive activity (Stat1C), we examined the effect of persistent Stat1 activity on lung cancer cell growth, survival and gene expression. We identified no significant effect of Stat1C alone or with interferon-gamma (IFN-gamma) on lung cancer cell growth or survival. Consistent with prior reports, Stat1C expression alone elicited minimal changes in gene expression and required costimulatory IFN-gamma for full activity. Using oligonucleotide gene arrays and quantitative real-time PCR, we identified numerous proinflammatory gene products and chemokines regulated by IFN-gamma/Stat1C signaling. These results suggest the major role of IFN-gamma and Stat1 in lung cells is to direct a proinflammatory gene expression program rather than have major effects on cell growth or survival or both.
Authors:
Jiannong Li; Bin Yu; Lanxi Song; Steve Eschrich; Eric B Haura
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research     Volume:  27     ISSN:  1079-9907     ISO Abbreviation:  J. Interferon Cytokine Res.     Publication Date:  2007 Mar 
Date Detail:
Created Date:  2007-03-12     Completed Date:  2007-05-01     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  9507088     Medline TA:  J Interferon Cytokine Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  209-20     Citation Subset:  IM    
Affiliation:
Thoracic Oncology/Experimental Therapeutics, H. Lee Moffitt Cancer Center and Research Institute, Tampa, FL 33612, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Antiviral Agents / pharmacology*
Carcinoma, Non-Small-Cell Lung / genetics,  metabolism*
Cell Line, Tumor
Cell Proliferation / drug effects
Cell Survival / drug effects,  genetics
Chemokines / biosynthesis,  genetics
Gene Expression Regulation, Neoplastic / drug effects*,  genetics
Humans
Inflammation / genetics,  metabolism
Interferon-gamma / pharmacology*
STAT1 Transcription Factor / genetics,  metabolism*
STAT3 Transcription Factor / genetics,  metabolism
Signal Transduction / drug effects*,  genetics
Chemical
Reg. No./Substance:
0/Antiviral Agents; 0/Chemokines; 0/STAT1 Transcription Factor; 0/STAT1 protein, human; 0/STAT3 Transcription Factor; 0/STAT3 protein, human; 82115-62-6/Interferon-gamma

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