Document Detail


Effects of blood glutamate scavenging on cortical evoked potentials.
MedLine Citation:
PMID:  20607387     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
It is well known that traumatic or ischemic brain injury is followed by acute excitotoxicity caused by the presence of abnormally high glutamate (Glu) in brain fluids. It has recently been demonstrated that excess Glu can be eliminated from brain into blood following the intravenous administration of oxaloacetate (OxAc), which, by scavenging blood Glu, induces an enhanced and neuroprotective brain-to-blood Glu efflux. In this study, we subjected rats to intravenous OxAc administration (i.v., 12.5, 25, and 50 mg/kg, respectively), and studied its effects on somatosensory evoked cortical potentials (EPs). Against our expectation, the amplitudes of EPs did not decrease but increased in a dose- and time-dependent manner after OxAc administration. Similar effects were observed when blood Glu scavenging was enhanced by combining OxAc (12.5 mg/kgbw) with recombinant glutamate-oxaloacetate transaminase (GOT, 0.14 nmol/100 g rat). On the basis of these results, we suggest that the changes of amplitudes of the EPs involve not only a glutamatergic but also the weakening of a GABAergic component. We cannot rule out the possibility that OxAc penetrates into the brain and improves mitochondrial functions.
Authors:
Dávid Nagy; Levente Knapp; Máté Marosi; Tamás Farkas; Zsolt Kis; László Vécsei; Vivian I Teichberg; József Toldi
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-07-06
Journal Detail:
Title:  Cellular and molecular neurobiology     Volume:  30     ISSN:  1573-6830     ISO Abbreviation:  Cell. Mol. Neurobiol.     Publication Date:  2010 Oct 
Date Detail:
Created Date:  2010-10-04     Completed Date:  2011-01-31     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8200709     Medline TA:  Cell Mol Neurobiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1101-6     Citation Subset:  IM    
Affiliation:
Department of Physiology, Anatomy and Neuroscience, University of Szeged, Közép fasor 52, Szeged, Hungary.
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MeSH Terms
Descriptor/Qualifier:
Animals
Aspartate Aminotransferase, Cytoplasmic / metabolism
Brain / drug effects,  metabolism*
Evoked Potentials, Somatosensory / drug effects,  physiology*
Glutamic Acid / blood*
Hydrogen-Ion Concentration
Male
Oxaloacetate / pharmacology
Rats
Rats, Wistar
Chemical
Reg. No./Substance:
0/Oxaloacetate; 56-86-0/Glutamic Acid; EC 2.6.1.-/Aspartate Aminotransferase, Cytoplasmic

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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