Document Detail


Effect of temperature on myocardial infarction in swine.
MedLine Citation:
PMID:  8967356     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Body core temperature in the normothermic range alters infarct size in rabbits. Moreover, temperature may modulate the protection by adenosine during a coronary artery occlusion. We investigated the effect of core temperature within the normothermic range (35-39 degrees C) on myocardial infarct size produced by a 45-min coronary occlusion in open-chest swine (n = 10), and we determined whether adenosine blockade with 8-phenyltheophylline and adenosine deaminase increased infarct size in the normothermic range (n = 9). After 4 h of reperfusion the area at risk and infarct size were determined with Evans blue dye and triphenyltetrazolium chloride. Infarct size strongly correlated with temperature (r2 = 0.71, P = 0.0001) so that at 35 degrees C no infarction occurred and with each 1 degree C increase in temperature 20% of the area at risk became infarcted. In contrast, neither the low levels of collateral flow (0.03 +/- 0.01 ml.min-1.g-1) nor the rate-pressure product correlated with infarct size. In the normothermic range, adenosine blockade had no effect on infarct size. The data demonstrate that temperature can exert a profound effect on infarct size but fail to demonstrate a protective effect on endogenous adenosine at normothermic temperatures. Our findings emphasize the need for stringent control of core temperature during investigation of interventions aimed at reducing infarct size.
Authors:
D J Duncker; C L Klassen; Y Ishibashi; S H Herrlinger; T J Pavek; R J Bache
Related Documents :
11940796 - Preconditioning and postresuscitation injury.
1415616 - Ischemic preconditioning attenuates acidosis and postischemic dysfunction in isolated r...
12871686 - Evidence of pharmacologic preconditioning during ptca by intravenous pretreatment with ...
11744026 - The effect of preconditioning (ischemic and pharmacological) on myocardial necrosis fol...
17458056 - Periodontitis as a risk factor of coronary heart diseases?
9307456 - Surgical revascularization for acute coronary insufficiency: analysis of risk factors f...
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  The American journal of physiology     Volume:  270     ISSN:  0002-9513     ISO Abbreviation:  Am. J. Physiol.     Publication Date:  1996 Apr 
Date Detail:
Created Date:  1996-12-04     Completed Date:  1996-12-04     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  0370511     Medline TA:  Am J Physiol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  H1189-99     Citation Subset:  IM    
Affiliation:
Department of Medicine, University of Minnesota Medical School, Minneapolis 55455, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Adenosine / antagonists & inhibitors
Adenosine Deaminase / pharmacology
Animals
Body Temperature*
Coronary Circulation
Female
Heart / physiopathology
Hemodynamics
Male
Myocardial Infarction / pathology*,  physiopathology
Theophylline / analogs & derivatives,  pharmacology
Grant Support
ID/Acronym/Agency:
HL-21872/HL/NHLBI NIH HHS; HL-32427/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
58-55-9/Theophylline; 58-61-7/Adenosine; 961-45-5/8-phenyltheophylline; EC 3.5.4.4/Adenosine Deaminase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Nitric oxide synthase does not participate in negative inotropic effect of acetylcholine in frog hea...
Next Document:  Relationship of renin-angiotensin system and ET-1 system activation in long-lasting response to post...