Document Detail


Effect of recombinant Panton-Valentine leukocidin in vitro on apoptosis and cytokine production of human alveolar macrophages.
MedLine Citation:
PMID:  20453909     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Panton-Valentine leukocidin (PVL) is associated with rare cases of necrotizing pneumonia that occur in otherwise healthy individuals. Human alveolar macrophages (HAMs) are major effector cells in host defense against infections. However, the impact of PVL on HAMs is uncertain. We evaluated the role of PVL in cytotoxicity and production of inflammatory cytokines secreted by HAMs. HAMs were purified from bronchoalveolar lavage fluid. Recombinant PVL (rPVL) was used in the study to interfere with HAM apoptosis and cytokine production in vitro. Hoechst 33342 fluorescence staining, transmission electron microscopy examination, and flow cytometry indicated that rPVL (10 nmol/L) treatment resulted in HAMs with markedly apoptotic characteristics, and HAMs treated with rPVL at 100 nmol/L showed clear indication of necrosis. A treatment of rPVL at 10 nmol/L elicited the secretion of IL-10 by HAMs relative to untreated control cells, but there was a slight decrease in the constitutive secretion of tumor necrosis factor (TNF)-alpha. Our results indicate that PVL-treated samples decreased HAM viability, leading to apoptosis at low concentrations and necrosis at high concentrations. In addition, PVL-treated cells released increased amounts of IL-10 and decreased amounts of TNF-alpha under apoptosis-inducing concentrations. Therefore, we speculated that PVL could play a negative role in HAM function at lower concentrations.
Authors:
Benquan Wu; Wenxian Zhang; Jing Huang; Hui Liu; Tiantuo Zhang
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Canadian journal of microbiology     Volume:  56     ISSN:  1480-3275     ISO Abbreviation:  Can. J. Microbiol.     Publication Date:  2010 Mar 
Date Detail:
Created Date:  2010-05-10     Completed Date:  2010-07-12     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0372707     Medline TA:  Can J Microbiol     Country:  Canada    
Other Details:
Languages:  eng     Pagination:  229-35     Citation Subset:  IM    
Affiliation:
Department of Internal Medicine, Third Affiliated Hospital of Sun Yat-Sen University, Guangzhou, P R China. zswbq@163.com
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MeSH Terms
Descriptor/Qualifier:
Apoptosis / drug effects*
Bacterial Toxins / genetics,  toxicity*
Benzimidazoles / metabolism
Bronchoalveolar Lavage Fluid / cytology
Cells, Cultured
Exotoxins / genetics,  toxicity*
Flow Cytometry
Gene Expression Regulation / drug effects
Humans
Interleukin-10 / metabolism*,  secretion
Leukocidins / genetics,  toxicity*
Macrophages, Alveolar / drug effects*,  metabolism,  ultrastructure
Microscopy, Electron, Transmission
Necrosis
Recombinant Proteins / genetics,  toxicity*
Tumor Necrosis Factor-alpha / metabolism*,  secretion
Chemical
Reg. No./Substance:
0/Bacterial Toxins; 0/Benzimidazoles; 0/Exotoxins; 0/Leukocidins; 0/Panton-Valentine leukocidin; 0/Recombinant Proteins; 0/Tumor Necrosis Factor-alpha; 130068-27-8/Interleukin-10; 23491-52-3/HOE 33342

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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