Document Detail


Effect of electrical tooth stimulation on blood flow, interstitial fluid pressure and substance P and CGRP-immunoreactive nerve fibers in the low compliant cat dental pulp.
MedLine Citation:
PMID:  7521929     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The effect of vasodilation on simultaneously measured interstitial fluid pressure (IFP, micropuncture) and blood flow (laser-Doppler) in the low compliant pulpal connective tissue was investigated in 10 cats. Vasodilation was induced by electrical stimulation of the tooth after pretreatment with the sympathetic blocker guanethidine. Visualization of the sensory neuropeptides calcitonin gene-related peptide (CGRP) and substance P (SP) was performed using immunocytochemistry. The study was designed to answer the following questions. (1) Does vasodilation promptly increase IFP in low compliant tissues? (2) Does an increase in IFP counteract the blood flow increase? (3) Does repeated electrical stimulation cause reduced staining of CGRP- and SP-immunoreactive nerve fibers in the dental pulp? Electrical stimulation resulted consistently in a nearly synchronous increase in both blood flow and IFP. IFP was nearly doubled, from 6.3 +/- 0.18 mm Hg in control to 11.7 +/- 0.44 mm Hg, whereas blood flow increased by 28%. However, despite continued vasodilation the IFP fell to control level, or even lower, within 1-5 min. The results indicate that the increased IFP will promote fluid absorption into the blood, counteracting a further IFP increase in low compliant tissues during vasodilation. Accordingly, transmural pressure is only transitorily reduced and compression of vessels does not take place. There was considerably less CGRP- and SP-immunoreactive fibers in the stimulated teeth than in the contralateral controls, suggesting that the vasodilation was caused by liberation of these sensory neuropeptides.
Authors:
K J Heyeraas; S Kim; W H Raab; M R Byers; M Liu
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Microvascular research     Volume:  47     ISSN:  0026-2862     ISO Abbreviation:  Microvasc. Res.     Publication Date:  1994 May 
Date Detail:
Created Date:  1994-10-11     Completed Date:  1994-10-11     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0165035     Medline TA:  Microvasc Res     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  329-43     Citation Subset:  IM    
Affiliation:
Department of Physiology, University of Bergen, Norway.
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MeSH Terms
Descriptor/Qualifier:
Animals
Blood Flow Velocity
Blood Pressure / physiology
Calcitonin Gene-Related Peptide / metabolism
Cats
Dental Pulp / blood supply*,  innervation,  physiology
Electric Stimulation
Extracellular Space / physiology
Female
Immunohistochemistry
Male
Nerve Fibers / metabolism
Pressure
Substance P / metabolism
Vasodilation / physiology
Grant Support
ID/Acronym/Agency:
DEO5159/DE/NIDCR NIH HHS; DEO5605/DE/NIDCR NIH HHS
Chemical
Reg. No./Substance:
33507-63-0/Substance P; 83652-28-2/Calcitonin Gene-Related Peptide

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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