Document Detail


Effect of edoxaban on markers of coagulation in venous and shed blood compared with fondaparinux.
MedLine Citation:
PMID:  21544313     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Edoxaban, an oral direct factor Xa (FXa) inhibitor, is in phase III clinical development for stroke prevention in atrial fibrillation and treatment of venous thromboembolism. The shed blood model allows for study of activated coagulation at a site of standardised tissue injury due to local release of tissue factor. The objective of this study was to evaluate the effect of three doses of edoxaban on markers of coagulation in shed and venous blood versus placebo and a standard prophylactic dose of fondaparinux. A total of 100 healthy male subjects were randomised to receive single doses of one of five treatments: subcutaneously administered fondaparinux 2.5 mg; orally administered edoxaban 30, 60, or 120 mg; or placebo. The primary objective was measurement of blood coagulation markers prothrombin fragment 1+2 (F1+2) and thrombin-antithrombin (TAT) complex, and platelet activation marker β-thromboglobulin (β-TG), in venous and shed blood. Secondary objectives included pharmacokinetics, shed blood volume, and safety of edoxaban. Single doses of edoxaban caused rapid and significant decreases of F1+2, TAT, and β-TG in the shed blood model, indicating inhibition of thrombin generation and platelet activation. Inhibition was significantly less for fondaparinux versus edoxaban. Baseline-corrected F1+2, TAT, and β-TG values demonstrated sustained inhibition up to 24 hours for shed blood in the edoxaban groups but no significant inhibition in venous blood. Overall, edoxaban treatments were well tolerated. In conclusion, single oral doses of edoxaban 30, 60, or 120 mg caused rapid and sustained inhibition of coagulation up to 24 hours in the shed blood model.
Authors:
M Wolzt; M M Samama; S Kapiotis; K Ogata; J Mendell; S Kunitada
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Publication Detail:
Type:  Comparative Study; Journal Article; Randomized Controlled Trial; Research Support, Non-U.S. Gov't     Date:  2011-05-05
Journal Detail:
Title:  Thrombosis and haemostasis     Volume:  105     ISSN:  0340-6245     ISO Abbreviation:  Thromb. Haemost.     Publication Date:  2011 Jun 
Date Detail:
Created Date:  2011-06-07     Completed Date:  2011-10-27     Revised Date:  2012-01-10    
Medline Journal Info:
Nlm Unique ID:  7608063     Medline TA:  Thromb Haemost     Country:  Germany    
Other Details:
Languages:  eng     Pagination:  1080-90     Citation Subset:  IM    
Affiliation:
Department of Clinical Pharmacology, Medical University of Vienna, Allgemeines Krankenhaus Wien, A-1090 Vienna, Austria. michael.wolzt@meduniwien.ac.at
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MeSH Terms
Descriptor/Qualifier:
Antithrombin III
Biological Markers / blood
Blood Coagulation* / drug effects
Clinical Protocols
Clinical Trials, Phase III as Topic
Drug Dosage Calculations*
Factor Xa / antagonists & inhibitors
Humans
Male
Peptide Fragments / blood
Peptide Hydrolases / blood
Platelet Activation / drug effects
Polysaccharides / administration & dosage,  adverse effects
Prothrombin
Pyridines / administration & dosage*,  adverse effects,  pharmacokinetics,  pharmacology
Thiazoles / administration & dosage*,  adverse effects,  pharmacokinetics,  pharmacology
beta-Thromboglobulin / metabolism
Chemical
Reg. No./Substance:
0/Biological Markers; 0/Peptide Fragments; 0/Polysaccharides; 0/Pyridines; 0/Thiazoles; 0/antithrombin III-protease complex; 0/beta-Thromboglobulin; 0/fondaparinux; 0/prothrombin fragment 1.2; 480449-70-5/edoxaban; 9000-94-6/Antithrombin III; 9001-26-7/Prothrombin; EC 3.4.-/Peptide Hydrolases; EC 3.4.21.6/Factor Xa

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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