Document Detail


Effect of chronic and selective endothelin receptor antagonism on microvascular function in type 2 diabetes.
MedLine Citation:
PMID:  18424628     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Vascular dysfunction, which presents either as an increased response to vasoconstrictors or an impaired relaxation to dilator agents, results in worsened cardiovascular outcomes in diabetes. We have established that the mesenteric circulation in Type 2 diabetes is hyperreactive to the potent vasoconstrictor endothelin-1 (ET-1) and displays increased nitric oxide-dependent vasodilation. The current study examined the individual and/or the relative roles of the ET receptors governing vascular function in the Goto-Kakizaki rat, a mildly hyperglycemic, normotensive, and nonobese model of Type 2 diabetes. Diabetic and control rats received an antagonist to either the ET type A (ETA; atrasentan; 5 mg x kg(-1) x day(-1)) or type B (ET(B); A-192621; 15 or 30 mg x kg(-1) x day(-1)) receptors for 4 wk. Third-order mesenteric arteries were isolated, and vascular function was assessed with a wire myograph. Maximum response to ET-1 was increased in diabetes and attenuated by ETA antagonism. ETB blockade with 15 mg/kg A-192621 augmented vasoconstriction in controls, whereas it had no further effect on ET-1 hyperreactivity in diabetes. The higher dose of A-192621 showed an ETA-like effect and decreased vasoconstriction in diabetes. Maximum relaxation to acetylcholine (ACh) was similar across groups and treatments. ETB antagonism at either dose had no effect on vasorelaxation in control rats, whereas in diabetes the dose-response curve to ACh was shifted to the right, indicating a decreased relaxation at 15 mg/kg A-192621. These results suggest that ETA receptor blockade attenuates vascular dysfunction and that ETB receptor antagonism exhibits differential effects depending on the dose of the antagonists and the disease state.
Authors:
Kamakshi Sachidanandam; Mostafa M Elgebaly; Alex K Harris; Jim R Hutchinson; Erin M Mezzetti; Vera Portik-Dobos; Adviye Ergul
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2008-04-18
Journal Detail:
Title:  American journal of physiology. Heart and circulatory physiology     Volume:  294     ISSN:  0363-6135     ISO Abbreviation:  Am. J. Physiol. Heart Circ. Physiol.     Publication Date:  2008 Jun 
Date Detail:
Created Date:  2008-06-09     Completed Date:  2008-08-15     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  100901228     Medline TA:  Am J Physiol Heart Circ Physiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  H2743-9     Citation Subset:  IM    
Affiliation:
Program in Clinical and Experimental Therapeutics, University of Georgia College of Pharmacy, Medical College of Georgia, Augusta, Georgia, USA.
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MeSH Terms
Descriptor/Qualifier:
Acetylcholine / pharmacology
Animals
Cardiovascular Agents / pharmacology*
Diabetes Mellitus, Type 2 / complications,  drug therapy*,  metabolism,  physiopathology
Diabetic Angiopathies / etiology,  metabolism,  physiopathology,  prevention & control*
Disease Models, Animal
Dose-Response Relationship, Drug
Endothelin-1 / metabolism
Male
Mesenteric Arteries / drug effects,  metabolism
Microcirculation / drug effects,  metabolism
Myography
Peptides, Cyclic / pharmacology
Pyrrolidines / pharmacology*
Rats
Rats, Wistar
Receptor, Endothelin A / antagonists & inhibitors*,  metabolism
Receptor, Endothelin B / antagonists & inhibitors*,  metabolism
Up-Regulation
Vasoconstriction / drug effects
Vasoconstrictor Agents / pharmacology
Vasodilation / drug effects
Vasodilator Agents / pharmacology
Viper Venoms / pharmacology
Grant Support
ID/Acronym/Agency:
DK-074385/DK/NIDDK NIH HHS; HL-076236/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/A 192621; 0/Cardiovascular Agents; 0/Endothelin-1; 0/Peptides, Cyclic; 0/Pyrrolidines; 0/Receptor, Endothelin A; 0/Receptor, Endothelin B; 0/Vasoconstrictor Agents; 0/Vasodilator Agents; 0/Viper Venoms; 0/atrasentan; 0/sarafotoxins s6; 136553-81-6/cyclo(Trp-Asp-Pro-Val-Leu); 51-84-3/Acetylcholine

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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