Document Detail


Effect of bee venom on transforming growth factor-beta1-treated hepatocytes.
MedLine Citation:
PMID:  20075187     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Bee venom (BV) has been used as treatment against a wide variety of ailments, including inflammatory diseases. Various studies have demonstrated anti-inflammatory and anticancer effects of BV. Transforming growth factor (TGF)-beta1 induces hepatocyte apoptosis via the mitochondrial permeability transition. However, there is no evidence or information regarding the antiapoptotic effect of BV on hepatocytes. The authors investigated the antiapoptotic effect of BV on TGF-beta1-treated hepatocytes. The results showed significant protection from DNA damage by BV treatment compared to corresponding TGF-beta1-treated hepatocytes without BV. BV suppressed TGF-beta1-induced activation of the bcl-2 family and caspase family of proteins, which resulted in inhibition of poly ADP-ribose polymerase (PARP) cleavage. Furthermore, BV is not cytotoxic in the low concentrations used in this study. Low concentrations of BV potently suppress the apoptotic response in TGF-beta1-treated hepatocytes; therefore, BV may have therapeutic potential for the treatment of liver diseases.
Authors:
Jung-Hyun Park; Kyung-Hyun Kim; Soo-Jung Kim; Woo-Ram Lee; Kwang-Gill Lee; Ji-Hyun Park; Kwan-Kyu Park
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  International journal of toxicology     Volume:  29     ISSN:  1092-874X     ISO Abbreviation:  Int. J. Toxicol.     Publication Date:    2010 Jan-Feb
Date Detail:
Created Date:  2010-01-15     Completed Date:  2010-03-23     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9708436     Medline TA:  Int J Toxicol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  49-56     Citation Subset:  IM    
Affiliation:
Department of Rehabilitation Medicine, Eulji University Hospital, Daejeon, South Korea.
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MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis / drug effects
Bee Venoms / pharmacology*
Cell Line
Cell Survival / drug effects
DNA Fragmentation / drug effects
Dose-Response Relationship, Drug
Formazans / metabolism
Hepatocytes / drug effects*,  metabolism,  pathology
Mice
Poly(ADP-ribose) Polymerases / antagonists & inhibitors,  drug effects
Protective Agents / pharmacology*
Proto-Oncogene Proteins c-bcl-2 / drug effects,  metabolism
Tetrazolium Salts / metabolism
Transforming Growth Factor beta / pharmacology*
Chemical
Reg. No./Substance:
0/Bee Venoms; 0/Formazans; 0/Protective Agents; 0/Proto-Oncogene Proteins c-bcl-2; 0/Tetrazolium Salts; 0/Transforming Growth Factor beta; 23305-68-2/MTT formazan; EC 2.4.2.30/Poly(ADP-ribose) Polymerases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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