Document Detail


Effect of ONO-3144, a free radical scavenger, on reoxygenation injury in anoxic myocardium.
MedLine Citation:
PMID:  2959083     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Thus, ONO-3144 treatment affords great protection against reoxygenation injury in the anoxic myocardium through the scavenging mechanism that includes .OH or closely related species of free radicals and also may inhibit thromboxane synthetase in the anoxic myocardium. We would hypothesize that free radical formation generated through the conversion of PGG2 to PGH2 should play an important role in the reoxygenation injury of the anoxic myocardium.
Authors:
H Kobayashi; K Kobayashi; Y Hirohata; K Aihara; T Ohtsuka
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Advances in prostaglandin, thromboxane, and leukotriene research     Volume:  17A     ISSN:  0732-8141     ISO Abbreviation:  Adv. Prostaglandin Thromboxane Leukot. Res.     Publication Date:  1987  
Date Detail:
Created Date:  1987-11-19     Completed Date:  1987-11-19     Revised Date:  2003-11-14    
Medline Journal Info:
Nlm Unique ID:  8211444     Medline TA:  Adv Prostaglandin Thromboxane Leukot Res     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  388-91     Citation Subset:  IM    
Affiliation:
Central Institute for Electron Microscopic Researches, Nippon Medical School, Tokyo, Japan.
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MeSH Terms
Descriptor/Qualifier:
Adenosine Triphosphate / metabolism
Animals
Anoxia / drug therapy*,  pathology,  physiopathology
Calcium / metabolism
Creatine Kinase / secretion
Free Radicals
Heart Diseases / drug therapy*,  pathology,  physiopathology
Propiophenones / pharmacology*
Rats
Chemical
Reg. No./Substance:
0/Free Radicals; 0/Propiophenones; 56-65-5/Adenosine Triphosphate; 7440-70-2/Calcium; 75060-92-3/2-aminomethyl-4-tert-butyl-6-propionylphenol; EC 2.7.3.2/Creatine Kinase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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