Document Detail


Effect of Helicobacter pylori eradication on gastric mucosal phospholipid content and its fatty acid composition.
MedLine Citation:
PMID:  11339417     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND AND AIMS: Whether Helicobacter pylori eradication alters gastric mucosal phospholipid contents and their fatty acid composition remains unclear. The aim of the present study was to clarify the effect of H. pylori eradication on gastric mucosal phosphatidylcholine (PC) content and its fatty acid composition. METHODS: Endoscopic biopsy specimens were taken from the antrum and body of each of 19 asymtomatic male volunteers for detection of H. pylori, histopathological assessment of gastritis, phospholipid determination and fatty acid analysis. All the subjects with H. pylori infection were treated with eradication therapy. Endoscopy and tissue sampling were repeated again 1 and 6 months after all treatment. RESULTS: In eight subjects, H. pylori infection was evident and was successfully eradicated. Pretreatment degrees of lymphocytes and plasma cells (inflammation) and polymorphonuclear neutrophils (activity) were greater in H. pylori-positive subjects compared with H. pylori-negative subjects (P<0.001), whereas the degree of inflammation decreased (P<0.001), and neutrophils had completely disappeared at 6 months after eradication. Moreover, the gastric mucosal PC contents at the antrum and body were unchanged within 1 month after cessation of treatment, but increased at 6 months after eradication (P<0.05). At 6 months after cessation of treatment, H. pylori-eradicated subjects had an increase (+30% at antrum, +18% at body) in linoleic acid composition and a decrease (-37%, -43%) in arachidonic acid composition of PC at the antrum and body, respectively. CONCLUSIONS: These findings suggest that H. pylori eradication reduces the production of various eicosanoids, resulting in the normalization of gastric mucosal PC content and its fatty acid composition, which may consequently cause the gastric mucosal hydrophobicity to be normalized.
Authors:
T Orihara; H Wakabayashi; A Nakaya; K Fukuta; S Makimoto; K Naganuma; A Entani; A Watanabe
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Journal of gastroenterology and hepatology     Volume:  16     ISSN:  0815-9319     ISO Abbreviation:  J. Gastroenterol. Hepatol.     Publication Date:  2001 Mar 
Date Detail:
Created Date:  2001-05-07     Completed Date:  2001-10-11     Revised Date:  2005-11-17    
Medline Journal Info:
Nlm Unique ID:  8607909     Medline TA:  J Gastroenterol Hepatol     Country:  Australia    
Other Details:
Languages:  eng     Pagination:  269-75     Citation Subset:  IM    
Affiliation:
Third Department of Internal Medicine, Faculty of Medicine, Toyama Medical and Pharmaceutical University, Sugitani, Japan.
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MeSH Terms
Descriptor/Qualifier:
Adult
Aged
Anti-Bacterial Agents*
Arachidonic Acid / metabolism
Drug Therapy, Combination / therapeutic use*
Fatty Acids / metabolism*
Gastric Mucosa / metabolism*,  pathology
Gastritis / microbiology,  pathology
Helicobacter Infections / drug therapy*,  metabolism*,  pathology
Helicobacter pylori* / isolation & purification
Humans
Linoleic Acid / metabolism
Male
Middle Aged
Phosphatidylcholines / metabolism
Stomach / microbiology
Chemical
Reg. No./Substance:
0/Anti-Bacterial Agents; 0/Fatty Acids; 0/Phosphatidylcholines; 2197-37-7/Linoleic Acid; 506-32-1/Arachidonic Acid

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