Document Detail


Effect of B-vitamin therapy on progression of diabetic nephropathy: a randomized controlled trial.
MedLine Citation:
PMID:  20424250     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
CONTEXT: Hyperhomocysteinemia is frequently observed in patients with diabetic nephropathy. B-vitamin therapy (folic acid, vitamin B(6), and vitamin B(12)) has been shown to lower the plasma concentration of homocysteine.
OBJECTIVE: To determine whether B-vitamin therapy can slow progression of diabetic nephropathy and prevent vascular complications.
DESIGN, SETTING, AND PARTICIPANTS: A multicenter, randomized, double-blind, placebo-controlled trial (Diabetic Intervention with Vitamins to Improve Nephropathy [DIVINe]) at 5 university medical centers in Canada conducted between May 2001 and July 2007 of 238 participants who had type 1 or 2 diabetes and a clinical diagnosis of diabetic nephropathy.
INTERVENTION: Single tablet of B vitamins containing folic acid (2.5 mg/d), vitamin B(6) (25 mg/d), and vitamin B(12) (1 mg/d), or matching placebo.
MAIN OUTCOME MEASURES: Change in radionuclide glomerular filtration rate (GFR) between baseline and 36 months. Secondary outcomes were dialysis and a composite of myocardial infarction, stroke, revascularization, and all-cause mortality. Plasma total homocysteine was also measured.
RESULTS: The mean (SD) follow-up during the trial was 31.9 (14.4) months. At 36 months, radionuclide GFR decreased by a mean (SE) of 16.5 (1.7) mL/min/1.73 m(2) in the B-vitamin group compared with 10.7 (1.7) mL/min/1.73 m(2) in the placebo group (mean difference, -5.8; 95% confidence interval [CI], -10.6 to -1.1; P = .02). There was no difference in requirement of dialysis (hazard ratio [HR], 1.1; 95% CI, 0.4-2.6; P = .88). The composite outcome occurred more often in the B-vitamin group (HR, 2.0; 95% CI, 1.0-4.0; P = .04). Plasma total homocysteine decreased by a mean (SE) of 2.2 (0.4) micromol/L at 36 months in the B-vitamin group compared with a mean (SE) increase of 2.6 (0.4) micromol/L in the placebo group (mean difference, -4.8; 95% CI, -6.1 to -3.7; P < .001, in favor of B vitamins).
CONCLUSION: Among patients with diabetic nephropathy, high doses of B vitamins compared with placebo resulted in a greater decrease in GFR and an increase in vascular events.
TRIAL REGISTRATION: isrctn.org Identifier: ISRCTN41332305.
Authors:
Andrew A House; Misha Eliasziw; Daniel C Cattran; David N Churchill; Matthew J Oliver; Adrian Fine; George K Dresser; J David Spence
Publication Detail:
Type:  Journal Article; Multicenter Study; Randomized Controlled Trial; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  JAMA : the journal of the American Medical Association     Volume:  303     ISSN:  1538-3598     ISO Abbreviation:  JAMA     Publication Date:  2010 Apr 
Date Detail:
Created Date:  2010-04-28     Completed Date:  2010-04-28     Revised Date:  2011-02-11    
Medline Journal Info:
Nlm Unique ID:  7501160     Medline TA:  JAMA     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1603-9     Citation Subset:  AIM; IM    
Affiliation:
Division of Nephrology, University of Western Ontario, London, Ontario, Canada.
Data Bank Information
Bank Name/Acc. No.:
ISRCTN/ISRCTN41332305
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MeSH Terms
Descriptor/Qualifier:
Aged
Diabetic Nephropathies / complications*,  drug therapy
Double-Blind Method
Female
Folic Acid / administration & dosage*,  adverse effects
Glomerular Filtration Rate
Humans
Hyperhomocysteinemia / drug therapy*,  etiology
Kidney / drug effects,  physiopathology
Male
Middle Aged
Vitamin B 12 / administration & dosage*,  adverse effects
Vitamin B 6 / administration & dosage*,  adverse effects
Vitamin B Complex / administration & dosage*,  adverse effects
Grant Support
ID/Acronym/Agency:
MCT-41551//Canadian Institutes of Health Research
Chemical
Reg. No./Substance:
12001-76-2/Vitamin B Complex; 59-30-3/Folic Acid; 68-19-9/Vitamin B 12; 8059-24-3/Vitamin B 6
Comments/Corrections
Comment In:
Evid Based Med. 2011 Feb;16(1):14-5   [PMID:  21047842 ]
Praxis (Bern 1994). 2010 Aug 11;99(16):987-8   [PMID:  20700876 ]
JAMA. 2010 Aug 11;304(6):636; author reply 636-7   [PMID:  20699451 ]
JAMA. 2010 Aug 11;304(6):636; author reply 636-7   [PMID:  20699452 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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