Document Detail


Effect of 100% O2 on passage of uncharged dextrans from blood to lung lymph.
MedLine Citation:
PMID:  2423496     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Since charge as well as size may influence the passage of plasma proteins from blood to lung lymph, we used uncharged dextrans as tracers to study the effects of hyperoxic lung injury on the molecular sieving properties of the pulmonary microcirculation in unanesthetized sheep. Polydisperse [3H]dextran was infused intravenously into five sheep before and after the animals breathed 100% O2 until lymph flow increased threefold (66-84 h). Lymph-to-plasma concentration ratios (L/P) were determined for [3H]dextran fractions of graded molecular sizes (1.6-8.4 nm effective radius) from samples obtained during the infusions. Before hyperoxia the blood-lymph barrier was highly restrictive to transport of [3H]dextrans above 5.0 nm in radius; steady-state L/P for these molecules averaged 0.03 or less. After the sheep breathed 100% O2, [3H]dextrans as large as 8.4 nm radius appeared in the lymph. Posthyperoxia, the L/P were significantly increased relative to prehyperoxia base-line values for every [3H]dextran fraction larger than 2.0 nm radius (P less than 0.05). In contrast, neither the L/P for albumin or total protein changed significantly. At autopsy, electron microscopy showed widespread damage to the endothelium of the alveolar capillaries with infrequent gaps between endothelial cells. In two control sheep, inhalation of compressed air for 96 h had no effect on lymph flow or L/P for the [3H]dextrans. We conclude that O2 poisoning reduced the selective sieving of uncharged dextrans across the blood-lymph barrier of the lungs and allowed larger dextrans to enter the lymph. These larger molecules may have leaked from the pulmonary microcirculation via disruptions in the continuity of the endothelial lining.
Authors:
J H Hansen-Flaschen; P N Lanken; G G Pietra; P M Sampson; L Johns; A P Fishman
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Journal of applied physiology (Bethesda, Md. : 1985)     Volume:  60     ISSN:  8750-7587     ISO Abbreviation:  J. Appl. Physiol.     Publication Date:  1986 May 
Date Detail:
Created Date:  1986-07-08     Completed Date:  1986-07-08     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  8502536     Medline TA:  J Appl Physiol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  1797-809     Citation Subset:  IM; S    
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MeSH Terms
Descriptor/Qualifier:
Animals
Capillary Permeability
Dextrans / blood,  metabolism*
Endothelium / metabolism
Lung / blood supply,  metabolism*
Lung Diseases / etiology*
Lung Injury
Lymph / metabolism*
Male
Microscopy, Electron
Oxygen / toxicity*
Pulmonary Edema / etiology,  pathology
Sheep
Grant Support
ID/Acronym/Agency:
HL-08805/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
7782-44-7/Oxygen; 9004-54-0/Dextrans

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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