Document Detail


Echocardiographic assessment of left ventricular hypertrophy in patients with obstructive or nonobstructive hypertrophic cardiomyopathy.
MedLine Citation:
PMID:  6686547     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
In patients with hypertrophic cardiomyopathy, wide-angle two-dimensional echocardiography is capable of detecting diverse patterns of myocardial hypertrophy that are often more extensive than may be appreciated by M-Mode echocardiography alone. In the vast majority of patients with hypertrophic cardiomyopathy the distribution of left ventricular hypertrophy is 'asymmetric'. Left ventricular wall thickening commonly involves substantial portions of the ventricular septum and free wall but rarely extends into the posterior segment of free wall (through which the M-mode beam passes). Four basic patterns of distribution of left ventricular hypertrophy may be identified by two-dimensional echocardiography in patients with hypertrophic cardiomyopathy. Most frequently (52% of patients) hypertrophy involves both the ventricular septum and anterolateral free wall (Type III). In other patients, hypertrophy is confined to the anterior portion of ventricular septum (Type I), involves the entire septum but not the free wall (Type II), or is limited to regions of the left ventricular wall other than the basal anterior ventricular septum (Type IV)--i.e. posterior segment of septum, anterolateral free wall, or septum in its apical one-half (apical hypertrophic cardiomyopathy). In patients with morphologic Type IV, the sites of left ventricular hypertrophy are inaccessible to the path of the conventional M-mode beam and the diagnosis of hypertrophic cardiomyopathy can only be established with two-dimensional echocardiography. Patients with the most marked and widespread hypertrophy involving the septum and free wall (Type III) more frequently demonstrated moderate to severe functional limitation, the pattern of left ventricular hypertrophy on electrocardiogram, and subaortic obstruction at rest produced by systolic anterior motion of the mitral valve in the presence of a small left ventricular outflow tract.
Authors:
B J Maron
Related Documents :
1877457 - Altered myocardial high-energy phosphate metabolites in patients with dilated cardiomyo...
3322687 - Dilated cardiomyopathy and myocarditis: natural history, etiology, clinical manifestati...
17212027 - Reversible cardiomyopathy.
19491077 - Recurrent takotsubo cardiomyopathy associated with pheochromocytoma.
2596417 - Surgical management of tachyarrhythmias.
8626947 - A randomized trial of low osmolar ionic versus nonionic contrast media in patients with...
Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  European heart journal     Volume:  4 Suppl F     ISSN:  0195-668X     ISO Abbreviation:  Eur. Heart J.     Publication Date:  1983 Nov 
Date Detail:
Created Date:  1984-03-22     Completed Date:  1984-03-22     Revised Date:  2004-11-17    
Medline Journal Info:
Nlm Unique ID:  8006263     Medline TA:  Eur Heart J     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  73-91     Citation Subset:  IM    
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Adolescent
Adult
Cardiomyopathy, Hypertrophic / diagnosis*,  physiopathology
Echocardiography*
Electrocardiography
Female
Heart Septum / physiopathology
Heart Ventricles / physiopathology
Humans
Mitral Valve / physiopathology
Myocardium / pathology

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  The acute and chronic effects of verapamil on left ventricular function in patients with hypertrophi...
Next Document:  Specificity of light and electron microscopic features of hypertrophic obstructive and nonobstructiv...