Document Detail


Early-onset sensorineural hearing loss is a prominent feature of H syndrome.
MedLine Citation:
PMID:  20399510     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
This case report describes two patients with H syndrome, a multisystemic autosomal recessive disorder, caused by mutations in the SLC29A3 gene. It is characterized by cutaneous hyperpigmentation, camptodactyly or flexion contractures and other features, among them hearing loss. The two patients had hearing loss as their presenting symptom, and had mutations in SLC29A3, one of them a novel mutation. The aim of this paper is to increase awareness to this recently described disorder, and to emphasize that H syndrome should be included in the differential diagnosis of congenital or acquired syndromic hearing loss in children.
Authors:
Yuval Ramot; Koji Sayama; Ruth Sheffer; Victoria Doviner; Nurith Hiller; Michal Kaufmann-Yehezkely; Abraham Zlotogorski
Publication Detail:
Type:  Case Reports; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  International journal of pediatric otorhinolaryngology     Volume:  74     ISSN:  1872-8464     ISO Abbreviation:  Int. J. Pediatr. Otorhinolaryngol.     Publication Date:  2010 Jul 
Date Detail:
Created Date:  2010-08-05     Completed Date:  2010-12-08     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8003603     Medline TA:  Int J Pediatr Otorhinolaryngol     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  825-7     Citation Subset:  IM    
Copyright Information:
Copyright 2010 Elsevier Ireland Ltd. All rights reserved.
Affiliation:
Department of Dermatology, Hadassah-Hebrew University Medical Center, Jerusalem, Israel.
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MeSH Terms
Descriptor/Qualifier:
Abnormalities, Multiple / genetics
Adolescent
Adult
Female
Hearing Loss, Sensorineural / genetics*
Humans
Hyperpigmentation / genetics
Male
Mutation*
Nucleoside Transport Proteins / genetics*
Syndrome
Chemical
Reg. No./Substance:
0/Nucleoside Transport Proteins; 0/SLC29A3 protein, human

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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