Document Detail


EF24, a novel synthetic curcumin analog, induces apoptosis in cancer cells via a redox-dependent mechanism.
MedLine Citation:
PMID:  15711178     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
In this study, we show that the novel synthetic curcumin analog, EF24, induces cell cycle arrest and apoptosis by means of a redox-dependent mechanism in MDA-MB-231 human breast cancer cells and DU-145 human prostate cancer cells. Cell cycle analysis demonstrated that EF24 causes a G2/M arrest in both cell lines, and that this cell cycle arrest is followed by the induction of apoptosis as evidenced by caspase-3 activation, phosphatidylserine externalization and an increased number of cells with a sub-G1 DNA fraction. In addition, we demonstrate that EF24 induces a depolarization of the mitochondrial membrane potential, suggesting that the compound may also induce apoptosis by altering mitochondrial function. EF24, like curcumin, serves as a Michael acceptor reacting with glutathione (GSH) and thioredoxin 1. Reaction of EF24 with these agents in vivo significantly reduced intracellular GSH as well as oxidized GSH in both the wild-type and Bcl-xL overexpressing HT29 human colon cancer cells. We therefore propose that the anticancer effect of a novel curcumin analog, EF24, is mediated in part by redox-mediated induction of apoptosis.
Authors:
Brian K Adams; Jiyang Cai; Jeff Armstrong; Marike Herold; Yang J Lu; Aiming Sun; James P Snyder; Dennis C Liotta; Dean P Jones; Mamoru Shoji
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Anti-cancer drugs     Volume:  16     ISSN:  0959-4973     ISO Abbreviation:  Anticancer Drugs     Publication Date:  2005 Mar 
Date Detail:
Created Date:  2005-02-15     Completed Date:  2005-05-24     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  9100823     Medline TA:  Anticancer Drugs     Country:  England    
Other Details:
Languages:  eng     Pagination:  263-75     Citation Subset:  IM    
Affiliation:
Program in Molecular and Systems Pharmacology, Emory University, Atlanta, GA 30322, USA.
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MeSH Terms
Descriptor/Qualifier:
Antineoplastic Agents / therapeutic use*
Apoptosis / drug effects*
Breast Neoplasms / drug therapy*,  metabolism
Caspase 3
Caspases / metabolism
Cell Cycle / drug effects
Colonic Neoplasms / drug therapy*,  metabolism
Curcumin / analogs & derivatives*
Female
Humans
Male
Oxidation-Reduction
Prostatic Neoplasms / drug therapy*,  metabolism
Reactive Oxygen Species / metabolism
Tumor Cells, Cultured
Grant Support
ID/Acronym/Agency:
CA82995/CA/NCI NIH HHS; ES 09047/ES/NIEHS NIH HHS
Chemical
Reg. No./Substance:
0/Antineoplastic Agents; 0/Reactive Oxygen Species; 458-37-7/Curcumin; EC 3.4.22.-/CASP3 protein, human; EC 3.4.22.-/Caspase 3; EC 3.4.22.-/Caspases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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