Document Detail


Dysregulation of leptin and testosterone production and their receptor expression in the human placenta with gestational diabetes mellitus.
MedLine Citation:
PMID:  20421132     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Whether the placenta contributes to some of the abnormal hormonal profiles in gestational diabetes mellitus (GDM) pregnancies and whether GDM affects placental endocrine signaling pathways are yet to be established. The objective of this study was to investigate differences in the expression of the placental steroid and peptide hormone synthesis-related factors, enzymes and their receptors between normal and GDM pregnancies. Nine term placentae from GDM pregnancies and twelve from healthy pregnancies were collected. The results of immunohistochemistry, Western blotting and semiquantitative RT-PCR indicated that mRNA and protein levels of leptin, leptin receptors, androgen receptor and FGF2 were significantly higher in the GDM placentae than non-GDM placentae; while NRIH3, NRIH2, StARD3, CYP11A1, HSD3B, HSD11B, HSD17B, ERalpha, ERbeta, progesterone receptor, FGF receptor-2, insulin receptor-alpha and -beta showed no differences. Interestingly, Western blotting and immunohistochemistry revealed that aromatase protein concentrations in the GDM placentae were significantly reduced without a change in mRNA levels. Moreover, androgen upregulated FGF2 expression in the placental villous explants. These findings suggest that the placentae of GDM pregnancies contribute to elevated testosterone and leptin levels due to a decrease in the conversion of testosterone to estrogens and to an increase in leptin production. The androgen and leptin signaling pathways may be over-activated by the presence of excessive ligands and overexpressed receptors in GDM placentae. Dysregulation of these two endocrine networks may contribute to placental abnormalities eventually increasing the frequency of maternal and fetal complications associated with GDM.
Authors:
P S Uzelac; X Li; J Lin; L D Neese; L Lin; S T Nakajima; H Bohler; Z Lei
Publication Detail:
Type:  Journal Article     Date:  2010-04-24
Journal Detail:
Title:  Placenta     Volume:  31     ISSN:  1532-3102     ISO Abbreviation:  Placenta     Publication Date:  2010 Jul 
Date Detail:
Created Date:  2010-07-08     Completed Date:  2010-10-04     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8006349     Medline TA:  Placenta     Country:  England    
Other Details:
Languages:  eng     Pagination:  581-8     Citation Subset:  IM    
Affiliation:
Division of Reproductive Endocrinology and Infertility, Department of Ob, Gyn and Women's Health, University of Louisville, Health Sciences Center, Louisville, KY 40292, USA.
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MeSH Terms
Descriptor/Qualifier:
Androgens / pharmacology
Diabetes, Gestational / metabolism*
Dihydrotestosterone / pharmacology
Female
Fibroblast Growth Factor 2 / biosynthesis
Humans
Leptin / biosynthesis*
Placenta / metabolism*
Pregnancy
RNA, Messenger / metabolism
Receptors, Androgen / biosynthesis
Receptors, Leptin / biosynthesis
Reverse Transcriptase Polymerase Chain Reaction
Testosterone / biosynthesis
Chemical
Reg. No./Substance:
0/Androgens; 0/Leptin; 0/RNA, Messenger; 0/Receptors, Androgen; 0/Receptors, Leptin; 103107-01-3/Fibroblast Growth Factor 2; 521-18-6/Dihydrotestosterone; 58-22-0/Testosterone

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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