Document Detail


Do MDL-1⁺ cells play a broad role in acute inflammation?
MedLine Citation:
PMID:  22005298     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Dengue virus (DV) reacts with myeloid DAP12-associating lectin-1 (MDL-1) on immature polymorphonuclear leukocytes. Interaction of DV with MDL-1+ cells triggers systemic inflammatory response syndrome (SIRS) and dengue shock syndrome (DSS), with subsequent multi-organ failure. In this issue of the JCI, Cheung et al. find that sterile acute liver injury in mice is associated with the accumulation of MDL-1+ cells and that triggering of these cells by DV or an MDL-1-specific agonist antibody leads to SIRS, shock, and death. These findings may have broad mechanistic and therapeutic implications for the development of SIRS, sepsis, and shock in humans exposed to a wide array of infectious and non-infectious conditions.
Authors:
Peter A Ward
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Publication Detail:
Type:  Comment; Journal Article; Research Support, N.I.H., Extramural     Date:  2011-10-17
Journal Detail:
Title:  The Journal of clinical investigation     Volume:  121     ISSN:  1558-8238     ISO Abbreviation:  J. Clin. Invest.     Publication Date:  2011 Nov 
Date Detail:
Created Date:  2011-11-02     Completed Date:  2012-01-03     Revised Date:  2012-02-07    
Medline Journal Info:
Nlm Unique ID:  7802877     Medline TA:  J Clin Invest     Country:  United States    
Other Details:
Languages:  eng     Pagination:  4234-7     Citation Subset:  AIM; IM    
Affiliation:
University of Michigan Medical School, Department of Pathology, Ann Arbor, Michigan, USA. pward@umich.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Female
Humans
Lectins, C-Type / immunology*
Myeloid Cells / immunology*
Receptors, Cell Surface / immunology*
Shock / etiology*,  immunology*
Grant Support
ID/Acronym/Agency:
GM-29507/GM/NIGMS NIH HHS; GM-61656/GM/NIGMS NIH HHS
Chemical
Reg. No./Substance:
0/Clec5a protein, mouse; 0/Lectins, C-Type; 0/Receptors, Cell Surface
Comments/Corrections
Comment On:
J Clin Invest. 2011 Nov;121(11):4446-61   [PMID:  22005300 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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