| Diverse molecular provocation of programmed cell death. | |
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MedLine Citation:
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PMID: 8918187 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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The experimental study and manipulation of programmed cell death has been greatly assisted by the identification of genetic and pharmacological tools that can either induce or block cell lethality. This review discusses new insights into the molecular sensing of perturbations induced by such tools, as well as the possible consequences of this detection in determining cell survival. |
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Authors:
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I E Wertz; M R Hanley |
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Publication Detail:
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Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. |
Journal Detail:
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Title: Trends in biochemical sciences Volume: 21 ISSN: 0968-0004 ISO Abbreviation: Trends Biochem. Sci. Publication Date: 1996 Oct |
Date Detail:
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Created Date: 1996-12-18 Completed Date: 1996-12-18 Revised Date: 2006-11-15 |
Medline Journal Info:
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Nlm Unique ID: 7610674 Medline TA: Trends Biochem Sci Country: ENGLAND |
Other Details:
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Languages: eng Pagination: 359-64 Citation Subset: IM |
Affiliation:
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Department of Biological Chemistry, School of Medicine, University of California at Davis 95616, USA. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Amyloid
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pharmacology Apoptosis / drug effects, genetics, physiology* Chelating Agents / pharmacology Cytoskeleton / chemistry DNA Damage / genetics Ion Channels / metabolism Oxidants / pharmacology Receptors, Cell Surface / metabolism Signal Transduction / genetics Tumor Necrosis Factor-alpha / pharmacology |
| Chemical | |
Reg. No./Substance:
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0/Amyloid; 0/Chelating Agents; 0/Ion Channels; 0/Oxidants; 0/Receptors, Cell Surface; 0/Tumor Necrosis Factor-alpha |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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