Document Detail


Dispersions of repolarization and ventricular arrhythmogenesis: lessons from animal models.
MedLine Citation:
PMID:  19027779     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Sudden cardiac death resulting from ventricular arrhythmogenesis is a leading cause of mortality in the developed world, accounting for up to 400,000 deaths per year in the US alone. Within the past forty years we have taken considerable leaps forward in our understanding of the causes and mechanisms underlying cardiac arrhythmias, particularly in the setting of inherited and acquired dysfunctions in ionic currents which constitute human long QT syndrome (LQTS). Impaired repolarization seen in LQTS commonly gives rise to an altered dispersion of repolarization, which is considered to provide the functional substrate necessary for the perpetuation of lethal arrhythmias. This review examines the bases for arrhythmias arising from repolarization heterogeneities and explores the applicability of the genetically amenable mouse for the study of arrhythmias arising from such mechanisms.
Authors:
Matthew J Killeen; Ian N Sabir; Andrew A Grace; Christopher L-H Huang
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review     Date:  2008-11-07
Journal Detail:
Title:  Progress in biophysics and molecular biology     Volume:  98     ISSN:  0079-6107     ISO Abbreviation:  Prog. Biophys. Mol. Biol.     Publication Date:    2008 Oct-Nov
Date Detail:
Created Date:  2009-03-16     Completed Date:  2009-05-11     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0401233     Medline TA:  Prog Biophys Mol Biol     Country:  England    
Other Details:
Languages:  eng     Pagination:  219-29     Citation Subset:  IM    
Affiliation:
Cardiovascular Research Centre, Massachusetts General Hospital, 149 13th Street, Charlestown, MA 02129, USA. matthew.killeen@gmail.com
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MeSH Terms
Descriptor/Qualifier:
Action Potentials
Animals
Arrhythmias, Cardiac / etiology,  physiopathology*
Death, Sudden, Cardiac / etiology
Disease Models, Animal
Dogs
Electrophysiological Phenomena
Heart Ventricles / physiopathology
Humans
Long QT Syndrome / congenital,  etiology,  physiopathology
Mice
Grant Support
ID/Acronym/Agency:
//British Heart Foundation; //Wellcome Trust

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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