Document Detail


Discovery of novel forkhead box O1 inhibitors for treating type 2 diabetes: improvement of fasting glycemia in diabetic db/db mice.
MedLine Citation:
PMID:  20736318     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Excessive hepatic glucose production through the gluconeogenesis pathway is partially responsible for the elevated glucose levels observed in patients with type 2 diabetes mellitus (T2DM). The forkhead transcription factor forkhead box O1 (Foxo1) plays a crucial role in mediating the effect of insulin on hepatic gluconeogenesis. Here, using a db/db mouse model, we demonstrate the effectiveness of Foxo1 inhibitor, an orally active small-molecule compound, as a therapeutic drug for treating T2DM. Using mass spectrometric affinity screening, we discovered a series of compounds that bind to Foxo1, identifying among them the compound, 5-amino-7-(cyclohexylamino)-1-ethyl-6-fluoro-4-oxo-1,4-dihydroquinoline-3-carboxylic acid (AS1842856), which potently inhibits human Foxo1 transactivation and reduces glucose production through the inhibition of glucose-6 phosphatase and phosphoenolpyruvate carboxykinase mRNA levels in a rat hepatic cell line. Oral administration of AS1842856 to diabetic db/db mice led to a drastic decrease in fasting plasma glucose level via the inhibition of hepatic gluconeogenic genes, whereas administration to normal mice had no effect on the fasting plasma glucose level. Treatment with AS1842856 also suppressed an increase in plasma glucose level caused by pyruvate injection in both normal and db/db mice. Taken together, these findings indicate that the Foxo1 inhibitor represents a new class of drugs for use in treating T2DM.
Authors:
Takeyuki Nagashima; Nobuharu Shigematsu; Riyo Maruki; Yasuharu Urano; Hirotsugu Tanaka; Akiyoshi Shimaya; Teruhiko Shimokawa; Masayuki Shibasaki
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Publication Detail:
Type:  Journal Article     Date:  2010-08-24
Journal Detail:
Title:  Molecular pharmacology     Volume:  78     ISSN:  1521-0111     ISO Abbreviation:  Mol. Pharmacol.     Publication Date:  2010 Nov 
Date Detail:
Created Date:  2010-10-20     Completed Date:  2010-12-07     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0035623     Medline TA:  Mol Pharmacol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  961-70     Citation Subset:  IM    
Affiliation:
Pharmacology Research Labs, Drug Discovery Research, Astellas Pharma Inc., Ibaraki, Japan. takeyuki.nagashima@jp.astellas.com
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Line, Tumor
Fasting
Forkhead Transcription Factors / antagonists & inhibitors*,  genetics
Glucose / biosynthesis
Glucose-6-Phosphatase / antagonists & inhibitors,  genetics
Humans
Hyperglycemia / drug therapy*,  metabolism
Hypoglycemic Agents / pharmacology*,  therapeutic use
Male
Mass Spectrometry
Mice
Nerve Tissue Proteins / antagonists & inhibitors
Phosphoenolpyruvate Carboxykinase (GTP) / antagonists & inhibitors,  genetics
Pyruvic Acid / pharmacology
Quinolones / pharmacology*,  therapeutic use
RNA, Messenger / antagonists & inhibitors
Rats
Structure-Activity Relationship
Transcriptional Activation
Chemical
Reg. No./Substance:
0/5-amino-7-(cyclohexylamino)-1-ethyl-6-fluoro-4-oxo-1,4-dihydroquinoline-3-carboxylic acid; 0/FOXO1 protein, human; 0/Forkhead Transcription Factors; 0/Foxo1 protein, mouse; 0/Hypoglycemic Agents; 0/Nerve Tissue Proteins; 0/Quinolones; 0/RNA, Messenger; 127-17-3/Pyruvic Acid; 147604-79-3/Foxo1 protein, rat; 50-99-7/Glucose; EC 3.1.3.9/Glucose-6-Phosphatase; EC 4.1.1.32/Phosphoenolpyruvate Carboxykinase (GTP)

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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