Document Detail


Differential repression by the transcription factor REST/NRSF of the various Ca2+ signalling mechanisms in pheochromocytoma PC12 cells.
MedLine Citation:
PMID:  20171735     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Expression of the nerve cell phenotype is orchestrated by the REST/NRSF transcription repressor, working on hundreds of genes recognized at a specific regulatory binding sequence. Most PC12 clones, the most frequently employed neuronal model, maintain low levels of REST; however a few, defective of neurosecretion, express high levels. To investigate the role of REST in Ca2+ signalling we studied the [Ca2+](i) changes in single cells of four clones, two wild-type and two defective, pre-treated for 5 days with NGF. We focused on Ca2+ influxes induced by depolarization and ATP. Only a subpopulation ( approximately 15%) of the defective, high REST cells responded to depolarization (Ca(V) expression approximately 10%). The ATP-induced intracellular Ca2+ release was little changed, whereas influx via ionotropic P2X receptors was decreased, in agreement with the decreased expression of P2X2 receptors. The percentage of defective cells expressing store-operated calcium entry (SOCE) following ATP stimulation was also lower. The responses of the defective clones were little affected by their differentiated state. In conclusion, our results revealed important new aspects of REST control of Ca2+ homeostasis, of potential physiological importance. The mechanisms of this control remain to be investigated.
Authors:
P Ariano; P Zamburlin; R D'Alessandro; J Meldolesi; D Lovisolo
Related Documents :
20197045 - The epidermal ca(2+) gradient: measurement using the phasor representation of fluoresce...
19255745 - (endo)cannabinoids mediate different ca2+ entry mechanisms in human bronchial epithelia...
19758695 - Calcineurin activation by slow calcium release from intracellular stores suppresses pro...
16938295 - 'quantal' ca(2+) release reassessed--a clue to oscillation and synchronization.
9851975 - Defective potassium currents in ataxia telangiectasia fibroblasts.
17391665 - Selective inhibition of calcium influx by 2-aminoethoxydiphenyl borate.
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-02-19
Journal Detail:
Title:  Cell calcium     Volume:  47     ISSN:  1532-1991     ISO Abbreviation:  Cell Calcium     Publication Date:  2010 Apr 
Date Detail:
Created Date:  2010-04-05     Completed Date:  2010-09-07     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8006226     Medline TA:  Cell Calcium     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  360-8     Citation Subset:  IM    
Copyright Information:
2010 Elsevier Ltd. All rights reserved.
Affiliation:
Department of Animal and Human Biology, University of Turin, via Accademia Albertina 13, I-10123 and NIS Centre of Excellence, Turin, Italy.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Adenosine Triphosphate / metabolism
Animals
Calcium Signaling / physiology
Cell Differentiation
Enzyme Repression
Membrane Potentials
Nerve Growth Factor / metabolism
Neurons / physiology*
Neurosecretion / genetics
PC12 Cells
Pheochromocytoma / genetics,  metabolism*,  pathology
Rats
Receptors, Purinergic P2 / genetics,  metabolism*
Repressor Proteins / genetics,  metabolism*
TRPC Cation Channels / biosynthesis*,  genetics
Chemical
Reg. No./Substance:
0/RE1-silencing transcription factor; 0/Receptors, Purinergic P2; 0/Repressor Proteins; 0/TRPC Cation Channels; 0/purinergic P2X receptor; 56-65-5/Adenosine Triphosphate; 9061-61-4/Nerve Growth Factor

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Synthesis, characterization and biodegradation of functionalized amino acid-based poly(ester amide)s...
Next Document:  Arsenic trioxide and ascorbic acid demonstrate promising activity against primary human CLL cells in...