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Differential regulation by fucoidan of IFN-γ-induced NO production in glial cells and macrophages.
MedLine Citation:
PMID:  20830678     Owner:  NLM     Status:  In-Process    
Abstract/OtherAbstract:
Fucoidan has shown numerous biological actions; however, the molecular bases of these actions have being issued. We examined the effect of fucoidan on NO production induced by IFN-γ and the molecular mechanisms underlying these effects in two types of cells including glia (C6, BV-2) and macrophages (RAW264.7, peritoneal primary cells). Fucoidan affected IFN-γ-induced NO and/or iNOS expression both in macrophages and glial cells but in a contrast way. Our data showed that in C6 glioma cells both JAK/STAT and p38 signaling positively regulated IFN-γ-induced iNOS, which were inhibited by fucoidan. In contrast, in RAW264.7 cells JAK/STAT is a positive regulator whereas p38 is a negative regulator of NO/iNOS production. In RAW264.7 cells, fucoidan enhanced p38 activation and induced TNF-α production. We also confirmed the dual regulation of p38 in BV-2 microglia and primary peritoneal macrophages. From these results, we suggest that fucoidan affects not only IFN-γ-induced NO/iNOS production differently in brain and peritoneal macrophages due to the different roles of p38 but the effects on TNF-α production in the two cell types. These novel observations including selective and cell-type specific effects of fucoidan on IFN-γ-mediated signaling and iNOS expression raise the possibility that it alters the sensitivity of cells to the p38 activation.
Authors:
Hang Do; Nam-Sung Kang; Suhkneung Pyo; Timothy R Billiar; Eun-Hwa Sohn
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Journal of cellular biochemistry     Volume:  111     ISSN:  1097-4644     ISO Abbreviation:  J. Cell. Biochem.     Publication Date:  2010 Dec 
Date Detail:
Created Date:  2010-11-30     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8205768     Medline TA:  J Cell Biochem     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1337-45     Citation Subset:  IM    
Copyright Information:
Copyright © 2010 Wiley-Liss, Inc.
Affiliation:
Division of Immunopharmacology, College of Pharmacy, Sungkyunkwan University, Kyunggi-do 440-746, Republic of Korea.
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