Document Detail


Differential intra-abdominal adipose tissue profiling in obese, insulin-resistant women.
MedLine Citation:
PMID:  19711137     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: We recently identified differences in abdominal subcutaneous adipose tissue (SAT) from insulin-resistant (IR) as compared to obesity-matched insulin sensitive individuals, including accumulation of small adipose cells, decreased expression of cell differentiation markers, and increased inflammatory activity. This study was initiated to see if these changes in SAT of IR individuals were present in omental visceral adipose tissue (VAT); in this instance, individuals were chosen to be IR but varied in degree of adiposity. We compared cell size distribution and genetic markers in SAT and VAT of IR individuals undergoing bariatric surgery.
METHODS: Eleven obese/morbidly obese women were IR by the insulin suppression test. Adipose tissue surgical samples were fixed in osmium tetroxide for cell size analysis via Beckman Coulter Multisizer. Quantitative real-time polymerase chain reaction for genes related to adipocyte differentiation and inflammation was performed.
RESULTS: While proportion of small cells and expression of adipocyte differentiation genes did not differ between depots, inflammatory genes were upregulated in VAT. Diameter of SAT large cells correlated highly with increasing proportion of small cells in both SAT and VAT (r = 0.85, p = 0.001; r = 0.72, p = 0.01, respectively). No associations were observed between VAT large cells and cell size variables in either depot. The effect of body mass index (BMI) on any variables in both depots was negligible.
CONCLUSIONS: The major differential property of VAT of IR women is increased inflammatory activity, independent of BMI. The association of SAT adipocyte hypertrophy with hyperplasia in both depots suggests a primary role SAT may have in regulating regional fat storage.
Authors:
Alice Liu; Tracey McLaughlin; Teresa Liu; Arthur Sherman; Gail Yee; Fahim Abbasi; Cindy Lamendola; John Morton; Samuel W Cushman; Gerald M Reaven; Philip S Tsao
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Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural     Date:  2009-08-27
Journal Detail:
Title:  Obesity surgery     Volume:  19     ISSN:  1708-0428     ISO Abbreviation:  Obes Surg     Publication Date:  2009 Nov 
Date Detail:
Created Date:  2009-10-28     Completed Date:  2010-03-19     Revised Date:  2012-05-07    
Medline Journal Info:
Nlm Unique ID:  9106714     Medline TA:  Obes Surg     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1564-73     Citation Subset:  IM    
Affiliation:
Division of Endocrinology, Department of Medicine, Stanford University Medical Center, 300 Pasteur Drive Rm S-025, Stanford, CA 94305, USA. aliceliu@stanford.edu
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MeSH Terms
Descriptor/Qualifier:
Adipocytes / cytology,  physiology
Adipogenesis / physiology*
Adult
Body Mass Index
Cell Size
Female
Gene Expression
Humans
Immunohistochemistry
Inflammation / metabolism
Insulin Resistance*
Intra-Abdominal Fat / cytology*,  metabolism*
Middle Aged
Obesity / genetics,  metabolism*
Obesity, Morbid / genetics,  metabolism
Subcutaneous Fat, Abdominal / cytology*,  metabolism*
Young Adult
Grant Support
ID/Acronym/Agency:
1 R01 DK071309-01/DK/NIDDK NIH HHS; 5 F32 DK079578-02/DK/NIDDK NIH HHS; 5 R01 DK071333-04/DK/NIDDK NIH HHS; F32 DK079578-01/DK/NIDDK NIH HHS; F32 DK079578-02/DK/NIDDK NIH HHS; M01-RR00070/RR/NCRR NIH HHS; R01 DK080436-03/DK/NIDDK NIH HHS
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From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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