Document Detail


Differential effector pathways regulate memory CD8 T cell immunity against Plasmodium berghei versus P. yoelii sporozoites.
MedLine Citation:
PMID:  20097864     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Malaria results in >1,000,000 deaths per year worldwide. Although no licensed vaccine exists, much effort is currently focused on subunit vaccines that elicit CD8 T cell responses directed against Plasmodium parasite liver stage Ags. Multiple immune-effector molecules play a role in antimicrobial immunity mediated by memory CD8 T cells, including IFN-gamma, perforin, TRAIL, Fas ligand, and TNF-alpha. However, it is not known which pathways are required for memory CD8 T cell-mediated immunity against liver stage Plasmodium infection. In this study, we used a novel immunization strategy to generate memory CD8 T cells in the BALB/c mouse model of P. berghei or P. yoelii sporozoite infection to examine the role of immune-effector molecules in resistance to the liver stage infection. Our studies reveal that endogenous memory CD8 T cell-mediated protection against both parasite species is, in part, dependent on IFN-gamma, whereas perforin was only critical in protection against P. yoelii. We further show that neutralization of TNF-alpha in immunized mice markedly reduces memory CD8 T cell-mediated protection against both parasite species. Thus, our studies identify IFN-gamma and TNF-alpha as important components of the noncytolytic pathways that underlie memory CD8 T cell-mediated immunity against liver stage Plasmodium infection. Our studies also show that the effector pathways that memory CD8 T cells use to eliminate liver stage infection are, in part, Plasmodium species specific.
Authors:
Noah S Butler; Nathan W Schmidt; John T Harty
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-01-22
Journal Detail:
Title:  Journal of immunology (Baltimore, Md. : 1950)     Volume:  184     ISSN:  1550-6606     ISO Abbreviation:  J. Immunol.     Publication Date:  2010 Mar 
Date Detail:
Created Date:  2010-02-18     Completed Date:  2010-05-04     Revised Date:  2011-07-25    
Medline Journal Info:
Nlm Unique ID:  2985117R     Medline TA:  J Immunol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  2528-38     Citation Subset:  AIM; IM    
Affiliation:
Department of Microbiology, University of Iowa, Iowa City, IA 52242, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
CD8-Positive T-Lymphocytes / immunology*,  metabolism
Culicidae / parasitology
Dendritic Cells / immunology,  metabolism
Fas Ligand Protein / immunology,  metabolism
Female
Immunologic Memory / immunology
Interferon-gamma / deficiency,  genetics,  immunology
Listeria monocytogenes / immunology
Liver Diseases, Parasitic / immunology,  parasitology
Malaria / immunology*,  parasitology,  prevention & control
Malaria Vaccines / administration & dosage,  immunology*
Mice
Mice, Inbred BALB C
Mice, Knockout
Perforin / deficiency,  genetics,  immunology
Plasmodium berghei / immunology*
Plasmodium yoelii / immunology*
Signal Transduction / immunology
Sporozoites / immunology*
TNF-Related Apoptosis-Inducing Ligand / deficiency,  genetics,  immunology
Tumor Necrosis Factor-alpha / immunology,  metabolism
Grant Support
ID/Acronym/Agency:
R01 AI085515-01A1/AI/NIAID NIH HHS; R01 AI085515-02/AI/NIAID NIH HHS; T32 AI0726024/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/Fas Ligand Protein; 0/Fasl protein, mouse; 0/Malaria Vaccines; 0/TNF-Related Apoptosis-Inducing Ligand; 0/Tnfsf10 protein, mouse; 0/Tumor Necrosis Factor-alpha; 126465-35-8/Perforin; 82115-62-6/Interferon-gamma
Comments/Corrections

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