Document Detail


Differences between left and right ventricular anatomy determine the types of reentrant circuits induced by an external electric shock. A rabbit heart simulation study.
MedLine Citation:
PMID:  16055175     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Despite the fact that elucidating the mechanisms of cardiac vulnerability to electric shocks is crucial to understanding why defibrillation shocks fail, important aspects of cardiac vulnerability remain unknown. This research utilizes a novel anatomically based bidomain finite-element model of the rabbit ventricles to investigate the effect of shock polarity reversal on the reentrant activity induced by an external defibrillation-strength shock in the paced ventricles. The specific goal of the study is to examine how differences between left and right ventricular chamber anatomy result in differences in the types of reentrant circuits established by the shock. Truncated exponential monophasic shocks of duration 8 ms were delivered via two external electrodes at various timings. Vulnerability grids were constructed for shocks of reversed polarity (referred to as RV- or LV- when either the RV or the LV electrode is a cathode). Our results demonstrate that reversing electrode polarity from RV- to LV- changes the dominant type of post-shock reentry: it is figure-of-eight for RV- and quatrefoil for LV- shocks. Differences in secondary types of post-shock arrhythmia also occur following shock polarity reversal. These effects of polarity reversal are primarily due to the fact that the LV wall is thicker than the RV, resulting in a post-shock excitable gap that is predominantly within the LV wall for RV- shocks and in the septum for LV- shocks.
Authors:
Blanca Rodríguez; James C Eason; Natalia Trayanova
Related Documents :
16563115 - Early invasive revascularisation for patients critically ill after acute myocardial inf...
19940765 - Comparison of early and long-term results of percutaneous coronary interventions in pat...
17462415 - Cardiogenic shock: collaboration between cardiac surgery and cardiology subspecialties ...
9406175 - Cardiac depression and cellular injury in hemorrhagic shock and reinfusion: role of fre...
22118005 - Qt dispersion is not increased in familial mediterranean fever.
9554765 - Randomised comparison of micro stent i with palmaz-schatz stent placement for the elect...
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2005-07-11
Journal Detail:
Title:  Progress in biophysics and molecular biology     Volume:  90     ISSN:  0079-6107     ISO Abbreviation:  Prog. Biophys. Mol. Biol.     Publication Date:    2006 Jan-Apr
Date Detail:
Created Date:  2005-12-05     Completed Date:  2006-04-28     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  0401233     Medline TA:  Prog Biophys Mol Biol     Country:  England    
Other Details:
Languages:  eng     Pagination:  399-413     Citation Subset:  IM    
Affiliation:
Department of Biomedical Engineering, Lindy Boggs Center, Tulane University, Suite 500, Tulane University, New Orleans, LA 70118, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Arrhythmias, Cardiac / physiopathology
Defibrillators
Heart Ventricles / anatomy & histology
Models, Cardiovascular*
Rabbits
Ventricular Function*
Grant Support
ID/Acronym/Agency:
HL063196/HL/NHLBI NIH HHS; HL067322/HL/NHLBI NIH HHS; P20 EB001432/EB/NIBIB NIH HHS

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Plasma TAFI and soluble CD40 ligand do not predict reperfusion following thrombolysis for acute myoc...
Next Document:  Surgical care of elderly women with ovarian cancer: a population-based perspective.