Document Detail


Dietary fish oil and curcumin combine to modulate colonic cytokinetics and gene expression in dextran sodium sulphate-treated mice.
MedLine Citation:
PMID:  21401974     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Both fish oil (FO) and curcumin have potential as anti-tumour and anti-inflammatory agents. To further explore their combined effects on dextran sodium sulphate (DSS)-induced colitis, C57BL/6 mice were randomised to four diets (2 × 2 design) differing in fatty acid content with or without curcumin supplementation (FO, FO+2 % curcumin, maize oil (control, MO) or MO+2 % curcumin). Mice were exposed to one or two cycles of DSS in the drinking-water to induce either acute or chronic intestinal inflammation, respectively. FO-fed mice exposed to the single-cycle DSS treatment exhibited the highest mortality (40 %, seventeen of forty-three) compared with MO with the lowest mortality (3 %, one of twenty-nine) (P = 0·0008). Addition of curcumin to MO increased (P = 0·003) mortality to 37 % compared with the control. Consistent with animal survival data, following the one- or two-cycle DSS treatment, both dietary FO and curcumin promoted mucosal injury/ulceration compared with MO. In contrast, compared with other diets, combined FO and curcumin feeding enhanced the resolution of chronic inflammation and suppressed (P < 0·05) a key inflammatory mediator, NF-κB, in the colon mucosa. Mucosal microarray analysis revealed that dietary FO, curcumin and FO plus curcumin combination differentially modulated the expression of genes induced by DSS treatment. These results suggest that dietary lipids and curcumin interact to regulate mucosal homeostasis and the resolution of chronic inflammation in the colon.
Authors:
Qian Jia; Ivan Ivanov; Zlatomir Z Zlatev; Robert C Alaniz; Brad R Weeks; Evelyn S Callaway; Jennifer S Goldsby; Laurie A Davidson; Yang-Yi Fan; Lan Zhou; Joanne R Lupton; David N McMurray; Robert S Chapkin
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Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.     Date:  2011-03-15
Journal Detail:
Title:  The British journal of nutrition     Volume:  106     ISSN:  1475-2662     ISO Abbreviation:  Br. J. Nutr.     Publication Date:  2011 Aug 
Date Detail:
Created Date:  2011-08-02     Completed Date:  2011-09-29     Revised Date:  2012-01-04    
Medline Journal Info:
Nlm Unique ID:  0372547     Medline TA:  Br J Nutr     Country:  England    
Other Details:
Languages:  eng     Pagination:  519-29     Citation Subset:  IM    
Affiliation:
Program in Integrative Nutrition and Complex Diseases, Texas A&M University, College Station, TX, USA.
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MeSH Terms
Descriptor/Qualifier:
Acute Disease
Animals
Anti-Inflammatory Agents, Non-Steroidal / adverse effects,  therapeutic use
Chronic Disease
Colitis / diet therapy*,  immunology,  metabolism,  pathology
Colon / drug effects,  immunology,  metabolism*,  pathology
Curcumin / adverse effects,  therapeutic use*
Cytokines / genetics,  metabolism*
Dextran Sulfate / administration & dosage,  toxicity
Dietary Supplements*
Fish Oils / adverse effects,  therapeutic use*
Gene Expression Profiling
Gene Expression Regulation*
Intestinal Mucosa / drug effects,  immunology,  metabolism,  pathology
Irritants / administration & dosage,  toxicity
Male
Mice
Mice, Inbred C57BL
NF-kappa B / genetics,  metabolism
Oligonucleotide Array Sequence Analysis
Random Allocation
Survival Analysis
Grant Support
ID/Acronym/Agency:
CA129444/CA/NCI NIH HHS; CA59034/CA/NCI NIH HHS; DK071707/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Anti-Inflammatory Agents, Non-Steroidal; 0/Cytokines; 0/Fish Oils; 0/Irritants; 0/NF-kappa B; 458-37-7/Curcumin; 9042-14-2/Dextran Sulfate
Comments/Corrections
Comment In:
Br J Nutr. 2011 Dec;106(11):1763; author reply 1764   [PMID:  21933457 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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