Document Detail


Diet-induced changes in membrane gangliosides in rat intestinal mucosa, plasma and brain.
MedLine Citation:
PMID:  15795600     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
OBJECTIVES: The objective of this study was to determine if dietary gangliosides induce changes in the ganglioside content of intestinal mucosa, plasma and brain and to identify where GM3 and GD3 are localized in the enterocyte membrane. METHODS: Male 18-day-old Sprague-Dawley rats were fed a semipurified diet containing 20% (w/w) fat. The control diet contained triglyceride, reflecting the fat formulation of an existing infant formula. Two experimental diets were formulated by adding sphingomyelin (1% w/w of total fat) or a ganglioside-enriched lipid (0.1% w/w of total fat) to the control diet fat. The ganglioside fraction of ganglioside-enriched lipid diet contained more than 80% GD3. After 2 weeks of feeding, the total and individual ganglioside and cholesterol content was measured in small intestinal mucosa, plasma and brain. RESULTS: The ganglioside-enriched lipid diet significantly increased total gangliosides in the intestinal mucosa, plasma and brain compared with the control diet. The ganglioside-enriched lipid diet significantly increased the level of GD3 (7.5% w/w) in the intestine compared with control (3.2% w/w) while decreasing the level of GM3, the major ganglioside in the intestine. The ratio of cholesterol to ganglioside in the intestinal mucosa, plasma and brain decreased significantly in rats fed the ganglioside-enriched lipid diet compared with controls. Confocal microscopy showed that GM3 is exclusively localized in the apical membrane of the enterocyte whereas GD3 is primarily localized in the basolateral membrane. CONCLUSIONS:: The authors conclude that dietary ganglioside is absorbed in the small intestine and transported to different membrane sites, altering ganglioside levels in the intestinal mucosa, plasma and brain and thus possibly having the potential to change developing enterocyte function (and possibly that of other cell lines).
Authors:
Eek Joong Park; Miyoung Suh; Kal Ramanujam; Kurt Steiner; David Begg; M Thomas Clandinin
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of pediatric gastroenterology and nutrition     Volume:  40     ISSN:  0277-2116     ISO Abbreviation:  J. Pediatr. Gastroenterol. Nutr.     Publication Date:  2005 Apr 
Date Detail:
Created Date:  2005-03-29     Completed Date:  2005-09-13     Revised Date:  2010-11-02    
Medline Journal Info:
Nlm Unique ID:  8211545     Medline TA:  J Pediatr Gastroenterol Nutr     Country:  United States    
Other Details:
Languages:  eng     Pagination:  487-95     Citation Subset:  IM    
Affiliation:
Nutrition and Metabolism Research Group, Department of Agricultural, Food and Nutritional Science, University of Alberta, Edmonton, Alberta, Canada.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Brain Chemistry / drug effects*
Cholesterol / blood,  metabolism*
Enterocytes / cytology,  metabolism*
Fluorescent Antibody Technique
G(M3) Ganglioside / administration & dosage,  blood,  metabolism
Gangliosides / administration & dosage*,  blood,  metabolism*
Intestinal Absorption / drug effects
Intestinal Mucosa / cytology,  drug effects,  metabolism*
Male
Membrane Lipids / metabolism
Microscopy, Confocal
Random Allocation
Rats
Rats, Sprague-Dawley
Chemical
Reg. No./Substance:
0/G(M3) Ganglioside; 0/Gangliosides; 0/Membrane Lipids; 57-88-5/Cholesterol; 62010-37-1/ganglioside, GD3
Comments/Corrections
Erratum In:
J Pediatr Gastroenterol Nutr. 2010 Nov;51(5):687

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Mutation in the sterol 27-hydroxylase gene associated with fatal cholestasis in infancy.
Next Document:  Free amino acids in full-term and pre-term human milk and infant formula.