Document Detail


Dieckol rescues mice from lethal irradiation by accelerating hemopoiesis and curtailing immunosuppression.
MedLine Citation:
PMID:  20883140     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
PURPOSE: To evaluate the radioprotective effect of dieckol, a component of the brown algae Ecklonia cava. MATERIALS AND METHODS: Mice were lethally irradiated, treated with dieckol and surveyed for survival and the mechanism of radioprotection. RESULTS: Dieckol treatment lengthened the survival of irradiated mice and significantly accelerated the hemopoietic recovery of bone marrow cells and peripheral immune cells compared with irradiated, but untreated controls. Dieckol also enhanced the proliferation and differentiation of immune cells, which had been suppressed by ionising radiation. It turned out that the increased expression of oxygen radical-scavenging enzyme manganese superoxide dismutase (MnSOD) and subsequent reduction in DNA damage and lipid peroxidation was crucial for dieckol's radioprotective effect. In addition, the dieckol-induced modulation of protein 53 (p53)-dependent pathways further strengthened the radioresistance of immune cells. CONCLUSION: Dieckol can be a promising agent for reducing the time needed for reconstitution of hemopoietic cells after exposure to irradiation.
Authors:
Eunjin Park; Ginnae Ahn; Jin-Suk Yun; Min Ju Kim; So Jin Bing; Dae Seung Kim; Jehee Lee; Nam Ho Lee; Jae Woo Park; Youngheun Jee
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  International journal of radiation biology     Volume:  86     ISSN:  1362-3095     ISO Abbreviation:  Int. J. Radiat. Biol.     Publication Date:  2010 Oct 
Date Detail:
Created Date:  2010-10-04     Completed Date:  2010-11-01     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8809243     Medline TA:  Int J Radiat Biol     Country:  England    
Other Details:
Languages:  eng     Pagination:  848-59     Citation Subset:  IM; S    
Affiliation:
Jeju National University, South Korea.
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MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis / drug effects
Benzofurans / pharmacology*
Cell Proliferation / drug effects
DNA Damage
Free Radical Scavengers / metabolism
Gamma Rays
Gene Expression Regulation, Enzymologic / drug effects
Granulocyte-Macrophage Colony-Stimulating Factor / secretion
Hematopoiesis / drug effects*,  radiation effects
Immune Tolerance / drug effects*,  radiation effects
Immunosuppression
Kinetics
Lipid Peroxidation / drug effects
Mice
Radiation-Protective Agents / pharmacology*
Reactive Oxygen Species / metabolism
Signal Transduction / drug effects
Spleen / cytology,  drug effects
Survival Analysis
Tumor Suppressor Protein p53 / metabolism
Chemical
Reg. No./Substance:
0/Benzofurans; 0/Free Radical Scavengers; 0/Radiation-Protective Agents; 0/Reactive Oxygen Species; 0/Tumor Suppressor Protein p53; 0/dieckol; 83869-56-1/Granulocyte-Macrophage Colony-Stimulating Factor

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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