Document Detail


Diastolic coronary resistance and capacitance are independent of the duration of diastole.
MedLine Citation:
PMID:  2000989     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Systolic myocardial contraction may produce changes in coronary resistance and capacitance that persist throughout a normal diastole. In addition, coronary resistance and capacitance as determined in the arrested heart may not accurately describe normal diastolic behavior. To evaluate these possibilities, an identification method capable of characterizing the input impedance of the coronary circulation in as little as 150 ms was developed. Using this method, coronary dynamics were measured during early and late diastoles in the beating heart with tone intact as well as during adenosine-induced maximal vasodilation. Coronary dynamics were also measured in the arrested heart during maximal vasodilation. With vasomotor tone intact, the parameters of a lumped model of the coronary circulation showed no variation from early to late diastole. During maximal vasodilation, model parameters also showed no variation from early to late diastole. Parameters in the arrested heart were not statistically different from those of the beating heart during maximal vasodilation. However, model parameters determined during maximal vasodilation were significantly different from those determined with tone intact. These results suggest that although coronary resistance and capacitance are dependent on vasomotor tone, they remain constant throughout diastole and remain similar in the arrested heart.
Authors:
R M Judd; D A Redberg; R E Mates
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  The American journal of physiology     Volume:  260     ISSN:  0002-9513     ISO Abbreviation:  Am. J. Physiol.     Publication Date:  1991 Mar 
Date Detail:
Created Date:  1991-04-11     Completed Date:  1991-04-11     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0370511     Medline TA:  Am J Physiol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  H943-52     Citation Subset:  IM    
Affiliation:
Department of Mechanical and Aerospace Engineering, State University of New York, Buffalo 14260.
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MeSH Terms
Descriptor/Qualifier:
Animals
Blood Pressure
Coronary Circulation
Coronary Vessels / physiology*
Diastole*
Dogs
Female
Male
Models, Cardiovascular
Time Factors
Vascular Resistance*
Vasodilation
Grant Support
ID/Acronym/Agency:
1-KO8-HLB-01168/HL/NHLBI NIH HHS; 2-PO1-HLB-15194/HL/NHLBI NIH HHS

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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