Document Detail


Developmental perspectives on copy number abnormalities of the 22q11.2 region.
MedLine Citation:
PMID:  20497193     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The 22q11.2 chromosomal landscape predisposes to genomic rearrangements that are associated with a variety of clinical phenotypes. The most well known of these include the 22q11.2 deletion and Cat-eye syndromes (CES), but more recently other copy number abnormalities have been recognised, especially with increased use of microarrays in the investigation of patients with congenital malformations or cognitive impairment. In addition, mutations in the TBX1 gene have been found in patients with phenotypes reminiscent of 22q11.2 syndromes. Recent advances in our understanding of 22q11.2 genes and their interactions provide insight into the mechanisms underlying the phenotypic variability of the 22q11.2 syndromes, and suggest a possible common developmental pathway perturbed by copy number abnormalities of this locus.
Authors:
T Y Tan; C T Gordon; D J Amor; P G Farlie
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review     Date:  2010-04-23
Journal Detail:
Title:  Clinical genetics     Volume:  78     ISSN:  1399-0004     ISO Abbreviation:  Clin. Genet.     Publication Date:  2010 Sep 
Date Detail:
Created Date:  2010-08-10     Completed Date:  2011-02-02     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0253664     Medline TA:  Clin Genet     Country:  Denmark    
Other Details:
Languages:  eng     Pagination:  201-18     Citation Subset:  IM    
Affiliation:
Genetic Health Services Victoria, Royal Children's Hospital, Flemington Rd, Parkville, VIC 3052, Australia. tiong.tan@ghsv.org.au
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MeSH Terms
Descriptor/Qualifier:
Abnormalities, Multiple / genetics*,  pathology
Animals
Chromosome Aberrations*
Chromosomes, Human, Pair 22 / genetics*
Craniofacial Abnormalities
Gene Expression Regulation, Developmental
Humans
Mutation
Syndrome
T-Box Domain Proteins / genetics
Chemical
Reg. No./Substance:
0/T-Box Domain Proteins; 0/TBX1 protein, human

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