Document Detail


Determinants of cardiac fibrosis in experimental hypermineralocorticoid states.
MedLine Citation:
PMID:  7485478     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Uninephrectomized rats maintained on 1.0% NaCl to drink and infused with aldosterone (0.75 microgram/h) for 8 wk have previously been shown to develop hypertension, cardiac hypertrophy, and cardiac fibrosis. In the present study we have shown that K+ supplementation (1.0% NaCl plus 0.4% KCl drinking solution) alters neither the interstitial nor the perivascular fibrotic response to mineralocorticoid treatment. Second, rats receiving 0.75 microgram/h 9 alpha-fluorocortisol, a mineralocorticoid and glucocorticoid agonist, respond with hypertension and cardiac fibrosis without cardiac hypertrophy. Finally, intracerebroventricular infusion of the mineralocorticoid receptor antagonist RU-28318 blocks blood pressure elevation, but not cardiac hypertrophy or fibrosis, when aldosterone is coinfused peripherally. We conclude that the myocardial fibrosis observed in response to chronic mineralocorticoid elevation and salt loading is a humorally mediated event independent of hypokalemia, hypertension, and cardiac hypertrophy. It remains to be determined whether the fibrosis observed in the presence of excess salt represents a direct (e.g., cardiac) effect of mineralocorticoid hormones or one mediated via a primary action on classical epithelial aldosterone target tissues (e.g., kidney).
Authors:
M Young; G Head; J Funder
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  The American journal of physiology     Volume:  269     ISSN:  0002-9513     ISO Abbreviation:  Am. J. Physiol.     Publication Date:  1995 Oct 
Date Detail:
Created Date:  1995-12-12     Completed Date:  1995-12-12     Revised Date:  2003-11-14    
Medline Journal Info:
Nlm Unique ID:  0370511     Medline TA:  Am J Physiol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  E657-62     Citation Subset:  IM    
Affiliation:
Baker Medical Research Institute, Prahran, Victoria, Australia.
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MeSH Terms
Descriptor/Qualifier:
Adrenalectomy
Aldosterone / pharmacology
Aldosterone Antagonists / pharmacology
Animals
Desoxycorticosterone / pharmacology*
Drug Combinations
Fibrosis
Fludrocortisone / pharmacology
Heart / drug effects*
Hormone Antagonists / pharmacology
Male
Mifepristone / pharmacology
Myocardium / pathology*
Nephrectomy
Potassium / blood
Rats
Rats, Sprague-Dawley
Spironolactone / analogs & derivatives,  pharmacology
Chemical
Reg. No./Substance:
0/Aldosterone Antagonists; 0/Drug Combinations; 0/Hormone Antagonists; 127-31-1/Fludrocortisone; 52-01-7/Spironolactone; 52-39-1/Aldosterone; 64-85-7/Desoxycorticosterone; 7440-09-7/Potassium; 76676-34-1/RU 28318; 84371-65-3/Mifepristone

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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